Zobrazeno 1 - 10
of 59
pro vyhledávání: '"Kristiina, Heikinheimo"'
Autor:
Judith Pape, Deniz Bakkalci, Rawiya Al Hosni, Benjamin S Simpson, Kristiina Heikinheimo, Stefano Fedele, Umber Cheema
Publikováno v:
Journal of Tissue Engineering, Vol 13 (2022)
Ameloblastoma is a benign, locally invasive epithelial odontogenic neoplasm of the jaw. Treatment of choice is jaw resection, often resulting in significant morbidity. The aim of this study was to recapitulate ameloblastoma in a completely humanised
Externí odkaz:
https://doaj.org/article/0912d38704924aa687d75d318345cf66
Autor:
Deniz Bakkalci, Amir Zaki Abdullah Zubir, Syed Ali Khurram, Judith Pape, Kristiina Heikinheimo, Stefano Fedele, Umber Cheema
Publikováno v:
Matrix Biology Plus, Vol 16, Iss , Pp 100125- (2022)
Tumour development and progression is dependent upon tumour cell interaction with the tissue stroma. Bioengineering the tumour-stroma microenvironment (TME) into 3D biomimetic models is crucial to gain insight into tumour cell development and progres
Externí odkaz:
https://doaj.org/article/98aed28f53ae416fb0594835c6925e2d
Autor:
Santiago Peralta, Gerald E. Duhamel, William P. Katt, Kristiina Heikinheimo, Andrew D. Miller, Faraz Ahmed, Angela L. McCleary-Wheeler, Jennifer K. Grenier
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Abstract Ameloblastomas are odontogenic tumors that are rare in people but have a relatively high prevalence in dogs. Because canine acanthomatous ameloblastomas (CAA) have clinicopathologic and molecular features in common with human ameloblastomas
Externí odkaz:
https://doaj.org/article/d5c7128c160c48f0a26af1a3052b03de
Autor:
Judith Pape, Shinsuke Fujii, Javier Catón, Kristiina Heikinheimo, Satsuki Shidara, Shinji Matsumoto, Tatsufumi Fujimoto, Tamotsu Kiyoshima, Eijiro Jimi, Kari J Kurppa, Megumi Kokura, Takuma Ishibashi, Peter Morgan, Akira Kikuchi
Publikováno v:
The Journal of Pathology. 256:119-133
Ameloblastoma is an odontogenic neoplasm characterized by slow intraosseous growth with progressive jaw resorption. Recent reports have revealed that ameloblastoma harbours an oncogenic BRAFV600E mutation with mitogen-activated protein kinase (MAPK)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bd2faa41c0b18ad6c808c963f21667f6
https://doi.org/10.1002/path.5814
https://doi.org/10.1002/path.5814
Autor:
Angela L McCleary-Wheeler, Faraz Ahmed, Andrew D. Miller, William P. Katt, Gerald E. Duhamel, Santiago Peralta, Jennifer K. Grenier, Kristiina Heikinheimo
Publikováno v:
Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Scientific Reports, Vol 11, Iss 1, Pp 1-13 (2021)
Ameloblastomas are odontogenic tumors that are rare in people but have a relatively high prevalence in dogs. Because canine acanthomatous ameloblastomas (CAA) have clinicopathologic and molecular features in common with human ameloblastomas (AM), spo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9af490910bfceb210d5c9a17432881d9
http://europepmc.org/articles/PMC8423744
http://europepmc.org/articles/PMC8423744
Autor:
Judith Pape, Deniz Bakkalci, Rawiya Al Hosni, Benjamin S Simpson, Kristiina Heikinheimo, Stefano Fedele, Umber Cheema
Publikováno v:
Journal of Tissue Engineering. 13:204173142211405
Ameloblastoma is a benign, locally invasive epithelial odontogenic neoplasm of the jaw. Treatment of choice is jaw resection, often resulting in significant morbidity. The aim of this study was to recapitulate ameloblastoma in a completely humanised
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::aa990a28c21f040e1dd7fc1197243639
https://doi.org/10.1177/20417314221140500
https://doi.org/10.1177/20417314221140500
Autor:
J-M Huhtala, Heather Dawson, M Kovac, Camilla Kragelund, Gunnar Warfvinge, Daniel Baumhoer, Alexandra Thiel, Hannes Heikinheimo, Ari Ristimäki, Peter Morgan, Klaus Elenius, Kari J. Kurppa, Kristiina Heikinheimo
Publikováno v:
Journal of Dental Research. 98:54-60
BRAF V600E is the most common mutation in conventional ameloblastoma (AM) of the mandible. In contrast, maxillary AMs appear to harbor more frequently RAS, FGFR2, or SMO mutations. Unicystic ameloblastoma (UAM) is considered a less aggressive variant
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2be586df8dfbcf4e2eaf77c1740b9ded
https://doi.org/10.1177/0022034518798810
https://doi.org/10.1177/0022034518798810
Autor:
Santiago Peralta, Angela L. McCleary‐Wheeler, Gerald E. Duhamel, Kristiina Heikinheimo, Jennifer K. Grenier
Publikováno v:
Veterinary and Comparative Oncology. 17
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2869f099d6d7e11cc8e0f18b5bdc92bd
https://doi.org/10.1111/vco.12420
https://doi.org/10.1111/vco.12420
Autor:
Gerald E. Duhamel, Kristiina Heikinheimo, Angela L McCleary-Wheeler, Jennifer K. Grenier, Santiago Peralta
Publikováno v:
Veterinary and comparative oncology. 17(3)
Ameloblastoma is a locally aggressive odontogenic tumour that occurs in humans and dogs. Most ameloblastomas (AM) in humans harbour mutually-exclusive driving mutations in BRAF, HRAS, KRAS, NRAS or FGFR2 that activate MAPK signalling, and in SMO that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bc7a722ba0b180f5c092bc6ac53e6e03
https://pubmed.ncbi.nlm.nih.gov/31041834
https://pubmed.ncbi.nlm.nih.gov/31041834
Publikováno v:
International Journal of Oral and Maxillofacial Surgery, 46, 7, pp. 883-885
International Journal of Oral and Maxillofacial Surgery, 46, 883-885
International Journal of Oral and Maxillofacial Surgery, 46, 883-885
Item does not contain fulltext Periosteal fasciitis, considered a subtype of nodular fasciitis, is a rare benign soft tissue mass often misdiagnosed as a malignant lesion due to its fast and infiltrative growth pattern and histological features. Nodu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d55042b485c4dcf381485a5ceea340e5
https://doi.org/10.1016/j.ijom.2017.02.005
https://doi.org/10.1016/j.ijom.2017.02.005
