Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Kristen D. Yetming"'
Publikováno v:
mBio, Vol 15, Iss 6 (2024)
ABSTRACT Herpesvirus genomes are maintained as extrachromosomal plasmids within the nuclei of infected cells. Some herpesviruses persist within dividing cells, putting the viral genome at risk of being lost to the cytoplasm during mitosis because kar
Externí odkaz:
https://doaj.org/article/ca7d1d57b0204bf2ac83aad854c8857c
Publikováno v:
mBio, Vol 11, Iss 5 (2020)
ABSTRACT The genomes of DNA tumor viruses regain nuclear localization after nuclear envelope breakdown during mitosis through the action of a viral protein with a chromatin-tethering domain (CTD). Here, we report that the human cytomegalovirus (HCMV)
Externí odkaz:
https://doaj.org/article/0a07340b1ba840cd8eb8f8c1d7c83cff
Publikováno v:
mBio, Vol 11, Iss 5 (2020)
mBio
mBio
Human cytomegalovirus (HCMV) is the leading infectious cause of birth defects, represents a serious complication for immunocompromised HIV/AIDS and organ transplant patients, and contributes to both immunosenescence and cardiovascular diseases. HCMV
Autor:
Kristen D. Yetming, Lena N Lupey-Green, David J. Hughes, JJ L. Miranda, Sergei Biryukov, Jeffery T. Sample, Elessa M. Marendy
Publikováno v:
J Virol
Funding: U.S. Public Health Service grant AI110328 to J.T.S. and in part by Commonwealth Universal Research Enhancement (CURE) funds. L.N.L. received support from National Cancer Institute training grant T32 CA060395, and is a Lymphoma Research Found
Autor:
Kristen D. Yetming, Carol A. Dickerson, David J. Hughes, Elessa Marendy, Jeffery T. Sample, Clare E. Sample
Publikováno v:
Journal of Virology. 86:1034-1045
Establishment of persistent Epstein-Barr virus (EBV) infection requires transition from a program of full viral latency gene expression (latency III) to one that is highly restricted (latency I and 0) within memory B lymphocytes. It is well establish
Autor:
Kristen D. Yetming, David J. Spector
Publikováno v:
Virology. (2):171-177
The UL84 gene of human cytomegalovirus is implicated in the initiation of viral DNA replication during lytic infection. UL84 is essential for replication of a cloned viral origin of lytic replication (oriLyt) in vitro and mutants of strains AD169 or