Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Krishna Chatpar"'
Autor:
Alain Dagher, Samir Das, Melanie Legault, Rida Abou-Haidar, Clotilde Degroot, Annabel Seyller, Thomas M. Durcan, Henri Rabalais, Derek Lo, Stephanie O.M. Dyke, Viviane Poupon, Marie-Noëlle Boivin, Christine Rogers, Julien Doyon, Sonia Denise Lai Wing Sun, Krishna Chatpar, Guy A. Rouleau, Angela Genge, Zaliqa Rosli, Mahdieh Tabatabaei, Edward A. Fon, Jason Karamchandani, Alan C. Evans
Publikováno v:
Neuroinformatics. 20:139-153
In January 2016, the Montreal Neurological Institute-Hospital (The Neuro) declared itself an Open Science organization. This vision extends beyond efforts by individual scientists seeking to release individual datasets, software tools, or building pl
Autor:
Samir, Das, Rida, Abou-Haidar, Henri, Rabalais, Sonia Denise Lai Wing, Sun, Zaliqa, Rosli, Krishna, Chatpar, Marie-Noëlle, Boivin, Mahdieh, Tabatabaei, Christine, Rogers, Melanie, Legault, Derek, Lo, Clotilde, Degroot, Alain, Dagher, Stephanie O M, Dyke, Thomas M, Durcan, Annabel, Seyller, Julien, Doyon, Viviane, Poupon, Edward A, Fon, Angela, Genge, Guy A, Rouleau, Jason, Karamchandani, Alan C, Evans
Publikováno v:
Neuroinformatics. 20(1)
In January 2016, the Montreal Neurological Institute-Hospital (The Neuro) declared itself an Open Science organization. This vision extends beyond efforts by individual scientists seeking to release individual datasets, software tools, or building pl
Publikováno v:
Cell Death & Disease
Cell Death and Disease, Vol 9, Iss 5, Pp 1-14 (2018)
Cell Death and Disease, Vol 9, Iss 5, Pp 1-14 (2018)
Persistent vascular injury and degeneration in diabetes are attributed in part to defective reparatory function of angiogenic cells. Our recent work implicates endoplasmic reticulum (ER) stress in high-glucose-induced bone marrow (BM) progenitor dysf
Autor:
Krishna Chatpar, Vivian F. Irish, Chantal LeBlanc, Josefina Mendez, Yamile Lozano, Yannick Jacob, Fei Zhang
Publikováno v:
The Plant journal : for cell and molecular biology. 93(2)
Summary The CRISPR/Cas9 system has greatly improved our ability to engineer targeted mutations in eukaryotic genomes. While CRISPR/Cas9 appears to work universally, the efficiency of targeted mutagenesis and the adverse generation of off-target mutat