Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kourtney Sloan"'
Sex-specific trisomic Dyrk1a-related skeletal phenotypes during development in a Down syndrome model
Autor:
Jonathan M. LaCombe, Kourtney Sloan, Jared R. Thomas, Matthew P. Blackwell, Isabella Crawford, Flannery Bishop, Joseph M. Wallace, Randall J. Roper
Publikováno v:
Disease Models & Mechanisms, Vol 17, Iss 9 (2024)
Externí odkaz:
https://doaj.org/article/1572a482389742a1848b82c57d5c59f0
Autor:
Kourtney Sloan, Jared Thomas, Matthew Blackwell, Deanna Voisard, Eva Lana-Elola, Sheona Watson-Scales, Daniel L. Roper, Joseph M. Wallace, Elizabeth M. C. Fisher, Victor L. J. Tybulewicz, Randall J. Roper
Publikováno v:
Disease Models & Mechanisms, Vol 16, Iss 4 (2023)
Externí odkaz:
https://doaj.org/article/3bbbbb9016c543a4b1f805a022884fdf
Autor:
Raza Jamal, Jonathan LaCombe, Roshni Patel, Matthew Blackwell, Jared R. Thomas, Kourtney Sloan, Joseph M. Wallace, Randall J. Roper
Publikováno v:
PLoS ONE, Vol 17, Iss 2 (2022)
Bone abnormalities affect all individuals with Down syndrome (DS) and are linked to abnormal expression of DYRK1A, a gene found in three copies in people with DS and Ts65Dn DS model mice. Previous work in Ts65Dn male mice demonstrated that both genet
Externí odkaz:
https://doaj.org/article/c7c5b83ec4dd49aaaaa857bdbd911e6c
Publikováno v:
Genes, Vol 12, Iss 1729, p 1729 (2021)
Genes
Volume 12
Issue 11
Genes
Volume 12
Issue 11
Trisomy 21 (Ts21) causes alterations in skeletal development resulting in decreased bone mass, shortened stature and weaker bones in individuals with Down syndrome (DS). There is a sexual dimorphism in bone mineral density (BMD) deficits associated w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f481b117d9ea44d6704a1dcd6530616
https://www.mdpi.com/2073-4425/12/11/1729
https://www.mdpi.com/2073-4425/12/11/1729
Autor:
Raza Jamal, Jonathan LaCombe, Roshni Patel, Matthew Blackwell, Jared R. Thomas, Kourtney Sloan, Joseph M. Wallace, Randall J. Roper
Publikováno v:
PloS one. 17(2)
Bone abnormalities affect all individuals with Down syndrome (DS) and are linked to abnormal expression of DYRK1A, a gene found in three copies in people with DS and Ts65Dn DS model mice. Previous work in Ts65Dn male mice demonstrated that both genet
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::559694da8b51ef984ce30cba3b88b509
https://pubmed.ncbi.nlm.nih.gov/35196359
https://pubmed.ncbi.nlm.nih.gov/35196359