Zobrazeno 1 - 10
of 25
pro vyhledávání: '"Koray Gumustas"'
Publikováno v:
Turkish neurosurgery. 32(4)
AIM This study aimed to investigate the protective role of Hippophae rhamnoides L. (Sea buckthorn, SBT) in cold plus immobilization stress-induced oxidative and nitrosative stress in rats. MATERIAL AND METHODS Thirty-two Wistar albino rats were rando
Autor:
Taner Tanriverdi, Berna Yavuz, Fatma Ozlen, Tibet Kacira, Koray Gumustas, Pinar Atukeren, Rahsan Kemerdere, Hakan Hanimoglu, Bülent Canbaz
Publikováno v:
Neurological Research. 32:492-501
The aim of this study was to assess plasma and/or tissue levels of adhesion and apoptotic molecules, cytokines, nitric oxide metabolites, levels of lipid peroxidation, myeloperoxidase and superoxide dismutase in patients with glioblastoma multiforme
Autor:
İnci Alican, Meral Yüksel, Sevgin Özlem İşeri, Koray Gumustas, Yasemin Ersoy, Pinar Atukeren, Feriha Ercan
Publikováno v:
Journal of Gastroenterology and Hepatology. 24:1142-1148
Background and Aim: Sildenafil, a selective and potent inhibitor of cyclic guanosine monophosphate (cGMP)-specific phosphodiesterase (PDE)5, has a relaxant effect on the smooth muscle cells of the arterioles supplying the human corpus cavernosum acti
Autor:
Hakan Hnimoglu, Pinar Atukeren, Tibet Kacira, Galip Zihni Sanus, Bülent Canbaz, Rahsan Kemerdere, Taner Tanriverdi, Koray Gumustas, Mehmet Yasar Kaynar
Publikováno v:
Journal of Clinical Neuroscience. 15:1036-1042
Hypoxia-inducible factor-1 alpha (HIF-1alpha) is the major transcriptional factor involved in the adaptive response to hypoxia. The aim of this study was to assess HIF-1alpha in 22 patients with transitional meningioma (TM) and 26 patients with gliob
Autor:
Pinar Atukeren, Taner Tanriverdi, Hakan Hanimoglu, Koray Gumustas, Yusuf Tunali, Mehmet Yasar Kaynar, Galip Zihni Sanus, Sabri Aydin, Tibet Kacira
Publikováno v:
Clinical Neurology and Neurosurgery. 109:561-566
Purpose The purpose of this study was to assess oxidative DNA damage and total antioxidant capacity (TAC) in patients with transitional meningioma (TM) and to compare the results with normal brain tissues. Patients and methods Oxidative DNA damage an
Autor:
Bülent Canbaz, Koray Gumustas, Rahsan Kemerdere, Taner Tanriverdi, Tibet Kacira, Mehmet Yasar Kaynar, Hakan Hanimoglu, Galip Zihni Sanus, Pinar Atukeren
Publikováno v:
Journal of Cancer Research and Clinical Oncology. 133:627-633
The aim of this study was to assess glutathione peroxidase (GPx), glutathione reductase (GRx) and protein oxidation (POx) levels in patients with glioblastoma multiforme (GBM) and transitional meningioma (TM) and to compare with normal brain tissues.
Autor:
Bülent Canbaz, Hakan Hanimoglu, Saffet Tuzgen, Tibet Kacira, Reza Dashti, Taner Tanriverdi, Mehmet Yasar Kaynar, Pinar Atukeren, Koray Gumustas, Galip Zihni Sanus
Publikováno v:
Clinical Oncology. 19:177-181
Aims To assess oxidative DNA damage and total antioxidant capacity (TAC) in glioblastoma multiforme (GBM) and to compare the results with normal brain tissues. Materials and methods Oxidative DNA damage and TAC were evaluated in GBM tissues extracted
Autor:
Osman Özdemir, Sönmez Uydeş-Doğan, Koray Gumustas, Hande Özyoğurtçu, Özge Uyanik, F. İlkay Alp Yildirim, Aydın Gürel, Pinar Atukeren
Publikováno v:
Prostaglandinsother lipid mediators. 121
Statins are suggested to possess healing properties due to their antioxidant and antiinflammatory effects in animal ulcer models. In contrary, a clinical report indicated the formation of gastric ulcer by the use of atorvastatin. In this study, we ai
Publikováno v:
Neurological Research. 27:562-567
Experimental studies provide evidence that oxidative damage plays a role in the development of vasospasm after aneurysmal subarachnoid hemorrhage (SAH) but data from human studies is still limited. The purpose of this study was to investigate the tim
Autor:
Taner Tanriverdi, Necmettin Tanriover, Koray Gumustas, Ercan Tureci, Saffet Tuzgen, Cengiz Kuday, Mustafa Uzan
Publikováno v:
Neurological Research. 25:31-34
Nitric oxide (NO) is a potential mediator of secondary brain injury in the settings of cerebral ischemia and inflammation. Traumatic brain injury (TBI) alters the levels of stable end products of NO metabolism. We investigated these changes and attem