Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Kongping Li"'
Autor:
Haixia Wen, Luxi Li, Lixuan Zhan, Yunyan Zuo, Kongping Li, Meiqian Qiu, Heying Li, Weiwen Sun, En Xu
Publikováno v:
Cell Death and Disease, Vol 12, Iss 7, Pp 1-16 (2021)
Abstract Mitophagy alleviates neuronal damage after cerebral ischemia by selectively removing dysfunctional mitochondria. Phosphatase and tensin homolog (PTEN) induced putative kinase 1 (PINK1)/Parkin-mediated mitophagy is the most well-known type of
Externí odkaz:
https://doaj.org/article/def2083bf6b8442c817a6fdfa326096e
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Transplantation of bone marrow stromal cells (BMSCs) is a promising therapy for ischemic stroke. Previously, we had reported that the secondary degeneration occurred in the ipsilateral substantia nigra (SN) after permanent distal branch of middle cer
Externí odkaz:
https://doaj.org/article/ae98808a48cb43ff8309e5777a9714ea
Autor:
Lixuan Zhan, Yanyan Tang, Zhe Shi, En Xu, Weiwen Sun, Jiaqi Jiang, Linhui Peng, Kongping Li, Xialin Zuo
Publikováno v:
Antioxidantsredox signaling. 36(7-9)
Aims Rhodiola sacra is a widely-used pharmaceutical component with multiple functions, including anti-oxidation and anti-inflammation. However, the exact mechanisms involved in neuroprotection against transient global cerebral ischemia (tGCI) remains
Autor:
Dandan Liu, Weiwen Sun, Donghai Liang, Lixuan Zhan, Wei-Ping Liao, Kongping Li, Haixia Wen, Liu Liu, En Xu, Yanmei Wang, Baoxing Wu
Publikováno v:
Brain Pathology. 27:822-838
We previously reported that hypoxic postconditioning (HPC) ameliorated hippocampal neuronal death induced by transient global cerebral ischemia (tGCI) in adult rats. However, the mechanism of HPC-induced neuroprotection is still elusive. Notably, hea
Autor:
Lixuan Zhan, Wensheng Xu, Dandan Liu, Yafu Tan, Donghai Liang, Weiwen Sun, En Xu, Kongping Li, Liu Liu, Zhe Shi, Huarong Zhou
Publikováno v:
Frontiers in Molecular Neuroscience
Frontiers in Molecular Neuroscience, Vol 11 (0)
Frontiers in Molecular Neuroscience, Vol 11 (0)
Transient global cerebral ischemia (tGCI) causes excessive release of glutamate from neurons. Astrocytic glutamate transporter-1 (GLT-1) and glutamine synthetase (GS) together play a predominant role in maintaining glutamate at normal extracellular c
Autor:
Kongping Li, Liu Liu, Siyuan Chen, Weiwen Sun, Xinyong Zhu, Zhiwei Lu, En Xu, Donghai Liang, Lixuan Zhan
Publikováno v:
Cell Death & Disease
Autophagy disruption leads to neuronal damage in hypoxic–ischemic brain injury. Rab7, a member of the Rab GTPase superfamily, has a unique role in the regulation of autophagy. Hypoxic preconditioning (HPC) provides neuroprotection against transient