Zobrazeno 1 - 10
of 43
pro vyhledávání: '"Koh Arakawa"'
Autor:
Hiroaki Taniguchi, Nobukiyo Tanaka, Harumi Uto-Kondo, Haruo Nakamura, Ryuichi Kato, Fumitaka Ohsuzu, Yukihiko Momiyama, Koh Arakawa, Reiko Ohmori
Publikováno v:
Journal of Atherosclerosis and Thrombosis. 18:108-114
Resistin is an adipocytokine that may link inflammation and atherosclerosis.We studied the associations of resistin levels with cardiovascular events and restenosis.We measured pre-procedural serum resistin levels in 140 patients with coronary artery
Autor:
Hiroaki Taniguchi, Kazuhiro Ashida, Masayoshi Nagata, Haruo Nakamura, Yukihiko Momiyama, Akira Kameyama, Zahi A. Fayad, Kumiko Noya, Akiko Hara, Koh Arakawa, Reiko Ohmori, Teruyoshi Kihara, Fumitaka Ohsuzu
Publikováno v:
American Heart Journal. 148:137-143
Background Magnetic resonance imaging was recently reported to detect atherosclerotic plaques in thoracic and abdominal aortas. Methods Using magnetic resonance imaging, we investigated associations of risk factors and plasma inflammatory markers wit
Autor:
Haruo Nakamura, Fumio Ohsuzu, Shinji Tabata, Kenji Higashi, Kei Nakajima, Masatoshi Kusuhara, Koh Arakawa, Atsushi Yonemura, Reiko Ohmori, Takeshi Yamashita, Makoto Ayaori
Publikováno v:
Clinical Cardiology. 27:281-286
Background: Vascular endothelial growth factor (VEGF) has been implicated in both angiogenesis and ischemia. However, the relationship between plasma VEGF level and coronary artery disease remains unknown. Hypothesis: Plasma VEGF level may be associa
Publikováno v:
Clinical Cardiology. 26:31-35
Background: Oxidized low-density lipoprotein (oxLDL) not only plays an important role in plaque formation, but also impairs the endothelium-dependent relaxation. Constrictive remodeling rather than intimal hyperplasia mainly contributes to restenosis
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 22:1002-1007
Vulnerable plaque generally contains a thin fibrous cap, lipid pools, and reduced internal plaque collagen. Arterial fluorescence analysis can differentiate atherosclerotic lesions from normal arteries; however, the contribution of the lipid core to
Autor:
Kouji Miyazaki, Nobuo Hatori, Fumitaka Ohsuzu, Hiroshi Yoshizu, Tutomu Tanaka, Takanori Tahara, Tadashi Yamagishi, Toshio Shibuya, Koh Arakawa, Nobuyuki Masaki
Publikováno v:
Circulation Journal. 66:769-772
A 65-year-old man was admitted to the National Defense Medical College Hospital for acute anterolateral myocardial infarction and cardiogenic shock. Emergency coronary angiography demonstrated occlusion of the proximal left anterior descending artery
Autor:
Hirosi Yoshizu, Nobuo Hatori, Makoto Ayaori, Takanori Tahara, Kazuhiro Ashida, Toshio Shibuya, Koh Arakawa, Tadashi Yamagishi, Kouji Miyazaki, Fumitaka Ohsuzu, Tutomu Tanaka
Publikováno v:
Japanese Circulation Journal. 64:130-134
A 48-year-old man with a history of hypertension and diabetes mellitus was hospitalized with sudden onset of severe chest pain. He was in cardiogenic shock with a systolic pressure of 60 mm Hg. His electrocardiogram (ECG) showed ST-segment elevation
Autor:
Akira Kurita, Hirokuni Etsuda, Yoshihiro Matsushima, Koh Arakawa, Kimio Satomura, Kuhara R, Fumitaka Ohsuzu, Akimi Uehata, Akira Hamabe, Takasi Akima, Hiroyuki Kusano, Bonpei Takase
Publikováno v:
Clinical Cardiology. 22:417-421
Background and hypothesis: Brachial artery flow-mediated dilation (FMD), a noninvasive, widely used clinical index of endothelial function and magnitude of FMD, has been reported to be closely related to many coronary risk factors and coronary athero
Autor:
Nobukiyo Tanaka, Katsumi Hayashi, Kikuo Isoda, Koji Miyazaki, Masatoshi Kusuhara, Fumitaka Ohsuzu, Motoki Hara, Koh Arakawa, Kazuo Kimura
Publikováno v:
Journal of Echocardiography. 5:58-60
Coronary artery fistulas are uncommon entities with communication between the coronary artery and cardiac structure. We encounter a 70-year-old, asymptomatic woman with a large right coronary artery to left ventricle fistula. Non-invasive echocardiog
Publikováno v:
The Journal of Japan Atherosclerosis Society. 23:619-623