Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Kobe Masuoka"'
Autor:
Ivan M. Rosado-Mendez, Kevin K. Noguchi, Laura Castañeda-Martinez, George Kirvassilis, Sophie H. Wang, Francesca Manzella, Brant S. Swiney, Kobe Masuoka, Saverio Capuano, III, Kevin G. Brunner, Kristin Crosno, Quinton W. Guerrero, Hayley Whitson, Ansgar Brambrink, Heather S. Simmons, Andres F. Mejia, James A. Zagzebski, Timothy J. Hall, Chrysanthy Ikonomidou
Publikováno v:
Neurobiology of Disease, Vol 127, Iss , Pp 554-562 (2019)
Apoptosis is triggered in the developing mammalian brain by sedative, anesthetic or antiepileptic drugs during late gestation and early life. Whether human children are vulnerable to this toxicity mechanism remains unknown, as there are no imaging te
Externí odkaz:
https://doaj.org/article/f86792d81f6c469ab5499c822c67f712
Autor:
Chrysanthy Ikonomidou, George Kirvassilis, Brant S. Swiney, Sophie H. Wang, Jacob N. Huffman, Sasha L. Williams, Kobe Masuoka, Saverio Capuano, III, Kevin R. Brunner, Kristin Crosno, Heather S. Simmons, Andres F. Mejia, Christopher A. Turski, Ansgar Brambrink, Kevin K. Noguchi
Publikováno v:
Neurobiology of Disease, Vol 130, Iss , Pp 104489- (2019)
Sedatives and anesthetics can injure the developing brain. They cause apoptosis of neurons and oligodendrocytes, impair synaptic plasticity, inhibit neurogenesis and trigger long-term neurocognitive deficits. The projected vulnerable period in humans
Externí odkaz:
https://doaj.org/article/eb953d7fb22749b1bfc7e244b98ecfdf
Autor:
Saverio Capuano, Ansgar M. Brambrink, Kevin K. Noguchi, Kevin R. Brunner, Jacob Huffman, George Kirvassilis, Andres Mejia, Kobe Masuoka, Heather S. Simmons, Kristin Crosno, Brant S. Swiney, Sasha L. Williams, Sophie H. Wang, Chrysanthy Ikonomidou, Christopher A. Turski
Publikováno v:
Neurobiol Dis
Neurobiology of Disease, Vol 130, Iss, Pp 104489-(2019)
Neurobiology of Disease, Vol 130, Iss, Pp 104489-(2019)
Sedatives and anesthetics can injure the developing brain. They cause apoptosis of neurons and oligodendrocytes, impair synaptic plasticity, inhibit neurogenesis and trigger long-term neurocognitive deficits. The projected vulnerable period in humans