Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Ko Matsuo"'
Autor:
Taisuke Kitagawa, Kazuto Kobayashi, Takuya Yamanaka, Tomonori Ichikawa, Ko Matsuo, Takanori Sano, Masayoshi Yamasaki, Hiroshi Tanemura, Tomoki Ishigaki, Fumitaka Miya, Yutaka Naito
Publikováno v:
Japanese Journal of Stroke. 45:16-22
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b1662bcdc2d3177da47955bc9d1efde6
https://doi.org/10.3995/jstroke.11034
https://doi.org/10.3995/jstroke.11034
Autor:
Hirotaka Kida, Maki Umino, Yuichiro, Akira Taniguchi, Masayuki Maeda, Akihiro Shindo, Hidekazu Tomimoto, Atsushi Niwa, Masayuki Satoh, Hidehiro Ishikawa, Ai Ito, Ko Matsuo
Publikováno v:
Journal of Neurology. 266:1887-1896
The pathogenesis of cortical microinfarcts (CMIs) is considered to be heterogeneous including cerebral small vessel disease (SVD) such as hypertensive vasculopathy (HV) and cerebral amyloid angiopathy (CAA). Recent advances in MRI have enabled the de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::23aa2a11137470fbe9b2c46dd0a0d81b
https://doi.org/10.1007/s00415-019-09350-9
https://doi.org/10.1007/s00415-019-09350-9
Autor:
Masayoshi Yamasaki, Yutaka Naito, Takehiro Suzuki, Ko Matsuo, Kota Maekawa, Masunari Shibata, Toshiyuki Takahashi
Publikováno v:
Internal Medicine
Antibody against myelin oligodendrocyte glycoprotein (MOG-IgG) associated encephalitis is an important syndrome associated with MOG-IgG. However, there have been no reports of MOG-IgG-associated optic neuritis or demyelination following meningitis wi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::982bbbc2cbc4afd8f0b3ad402de4c066
https://doi.org/10.2169/internalmedicine.2845-19
https://doi.org/10.2169/internalmedicine.2845-19
Autor:
Akihiro Shindo, Hidekazu Tomimoto, Takakuni Maki, Atsushi Niwa, Takashi Ayaki, Ryosuke Takahashi, Yoshio Hashizume, Haruhiko Akiyama, Ko Matsuo, Ken-ichi Tabei, Nobukatsu Sawamoto, Hiroyasu Akatsu, Shinji Oikawa
Publikováno v:
Dementia and Geriatric Cognitive Disorders. 44:343-353
Background: Cerebral amyloid angiopathy (CAA) is classified as type 1 with capillary amyloid β (Aβ) or type 2 without capillary Aβ. While it is known that CAA activates complement, an inflammatory mediator, there is no information on the relations
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a71c0adcbe028dd1059e370e928880d9
https://doi.org/10.1159/000486091
https://doi.org/10.1159/000486091
Autor:
Yuichiro, Yoshio Hashizume, Akihiro Shindo, Masayuki Maeda, Hajime Sakuma, Hidekazu Tomimoto, Hidehiro Ishikawa, Ko Matsuo, Shinichi Takase, Atsushi Niwa, Hiroyasu Akatsu, Akira Taniguchi
Publikováno v:
Journal of Alzheimer's Disease
Microvascular lesions including cortical microinfarctions (CMIs) and cerebral lobar microbleeds (CMBs) are usually caused by cerebral amyloid angiopathy (CAA) in the elderly and are correlated with cognitive decline. However, their radiological-histo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ed9991698ff8bcea9b47513a27cb7d9
https://pubmed.ncbi.nlm.nih.gov/28697558
https://pubmed.ncbi.nlm.nih.gov/28697558
Autor:
Ai Itoh, Yukito Ueda, Ko Matsuo, Atsushi Niwa, Masayuki Satoh, Ken-ichi Tabei, Kana Matsuda, Keita Matsuura, Masaru Asahi, Hidekazu Tomimoto, Hidehiro Ishikawa, Akihiro Shindo
Publikováno v:
Journal of Neurology, Neurosurgery & Psychiatry. 90:957-958
Somatosensory functions are subdivided into two large groups: the elementary somatosensory functions, which consist of light touch, pain, thermal sensation, joint position sense and vibration sense, and the intermediate somatosensory functions, which
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d8937dbbffc3b976ec0380fef4edc03a
https://doi.org/10.1136/jnnp-2018-319174
https://doi.org/10.1136/jnnp-2018-319174
Publikováno v:
Neuropathology. 33:199-203
Over 100 mutations have been described in the presenilin-1 gene (PSEN1), resulting in familial Alzheimer disease (AD). However, of the limited number of autopsy cases, only one has been reported from an AD family with an L420R PSEN1 mutation. We desc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ebed90ed21aa3d74369a5a63a8865732
https://doi.org/10.1111/j.1440-1789.2012.01340.x
https://doi.org/10.1111/j.1440-1789.2012.01340.x
Autor:
Yuichiro, Akira Taniguchi, Hidekazu Tomimoto, Masayuki Maeda, Hirotaka Kida, Akihiro Shindo, Ko Matsuo
Publikováno v:
Journal of Neuroimaging. 23:28-32
BACKGROUND AND PURPOSE Cortical microinfarcts (CMIs) are detected as small foci restricted to the cerebral cortex in autopsy brains. CMIs are thought to be caused by cerebral amyloid angiopathy (CAA) in the elderly and may be a risk for dementia. We
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c3e09dd5bffd3ab5ed5a5ea9bb56a286
https://doi.org/10.1111/j.1552-6569.2012.00722.x
https://doi.org/10.1111/j.1552-6569.2012.00722.x
Autor:
Kenji Tadokoro, Tsuyoshi Tange, Mayumi Sato, N. Hashimoto, Ko Matsuo, Yoshinori Urano, Hideaki Oda, Hajime Kohama, Toyohiro Fukushima, Claude Jasmin, Junko Matsuoka
Publikováno v:
Pathology International. 41:259-264
Myeloproliferative syndrome was induced in adult DBA/2 mice by inoculation with myeloproliferative sarcoma virus (MPSV) and Friend murine leukemia virus (F-MuLV) as a helper virus. On day 26 after infection, the spleen weighed a maximum of 2.0 g (abo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b71aa86167bdafff1e5ff30929cda843
https://doi.org/10.1111/j.1440-1827.1991.tb03353.x
https://doi.org/10.1111/j.1440-1827.1991.tb03353.x
Autor:
Atsushi, Niwa, Ko, Matsuo, Akihiro, Shindo, Kenichiro, Yata, Taizo, Shiraishi, Hidekazu, Tomimoto
Publikováno v:
Neuropathology : official journal of the Japanese Society of Neuropathology. 33(2)
Over 100 mutations have been described in the presenilin-1 gene (PSEN1), resulting in familial Alzheimer disease (AD). However, of the limited number of autopsy cases, only one has been reported from an AD family with an L420R PSEN1 mutation. We desc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1ef6971af53f2ed61953bda91cce0738
https://pubmed.ncbi.nlm.nih.gov/22882713
https://pubmed.ncbi.nlm.nih.gov/22882713