Control of p21Cip by BRAP is critical for cardiomyocyte cell cycle progression and survival

Autor: Volland, C., Schott, P., Didié, M., Männer, J., Unsöld, B., Toischer, K., Schmidt, C., Urlaub, H., Nickels, K., Knöll, R., Schmidt, A., Guan, K., Hasenfuß, G., Seidler, T.

Publikováno v: Cardiovascular Research

AIMS: Identifying the key components in cardiomyocyte cell cycle regulation is of relevance for the understanding of cardiac development and adaptive and maladaptive processes in the adult myocardium. BRCA1-associated protein (BRAP) has been suggeste

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______1874::b48faa71fed211a0d82ed2e83d970407
https://hdl.handle.net/21.11116/0000-0005-C2BF-921.11116/0000-0004-3EDA-221.11116/0000-0005-C2C0-6

Correction to: The Sydney Heart Bank: improving translational research while eliminating or reducing the use of animal models of human heart disease (Biophysical Reviews, (2017), 9, 4, (431-441), 10.1007/s12551-017-0305-3)

Autor: dos Remedios, C.G., Lal, S. P., Li, A., McNamara, J., Keogh, A., Macdonald, P. S., Cooke, R., Ehler, E., Knöll, R., Marston, S. B., Stelzer, J., Granzier, H., Bezzina, C., van Dijk, S., De Man, F.S., Stienen, G. J.M., Odeberg, J., Pontén, F., Linke, W. A., van der Velden, J.

Publikováno v: Biophysical Reviews, 10(3). Springer Verlag
dos Remedios, C G, Lal, S P, Li, A, McNamara, J, Keogh, A, Macdonald, P S, Cooke, R, Ehler, E, Knöll, R, Marston, S B, Stelzer, J, Granzier, H, Bezzina, C, van Dijk, S, De Man, F S, Stienen, G J M, Odeberg, J, Pontén, F, Linke, W A & van der Velden, J 2018, ' Correction to : The Sydney Heart Bank: improving translational research while eliminating or reducing the use of animal models of human heart disease (Biophysical Reviews, (2017), 9, 4, (431-441), 10.1007/s12551-017-0305-3) ', Biophysical Reviews, vol. 10, no. 3 . https://doi.org/10.1007/s12551-018-0397-4

In the original version of this article, the name of one of the authors is not correct. The correct name should be W. A. Linke, which is shown correctly in the authorgroup section above.

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::5074555bd4116092c213356e98624aca
https://research.vumc.nl/en/publications/9da7c485-b203-498e-b917-5d8e3639fdfc

OTHER NMDs: EP.360 Is7 domain of titin is essential for cardiac function in mice

Autor: Biquand, A., Spinozzi, S., Tonino, P., Strom, J., Cosette, J., Elbeck, Z., Knöll, R., Granzier, H., Lostal, W., Richard, I.

Publikováno v: In Neuromuscular Disorders October 2021 31 Supplement 1:S159-S159

Myopalladin is upregulated in dilated cardiomyopathies patients and myopalladin knockout mice develop cardiac dilation and dysfunction following pressure overload

Autor: Filomena, M.C., Yamamoto, D.L., Carullo, P., Piroddi, N., Tesi, C., Scellini, B., Crispino, R., Zhang, J., Knöll, R., Polishchuk, R., Poggesi, C., Bang, M.L.

Publikováno v: In Journal of Molecular and Cellular Cardiology July 2018 120 Supplement:43-43

Towards a re-definition of 'cardiac hypertrophy' through a rational characterization of left ventricular phenotypes: a position paper of the Working Group 'Myocardial Function' of the ESC

Autor: Knöll, R., Iaccarino, Guido, Tarone, G., Hilfiker Kleiner, D., Bauersachs, J., Leite Moreira, A. F., Sugden, P. H., Balligand, J., E. S., Of

Many primary or secondary diseases of the myocardium are accompanied with complex remodelling of the cardiac tissue that results in increased heart mass, often identified as cardiac 'hypertrophy'. Although there have been numerous attempts at definin

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be6c7a82e7b63a38010438737d7ee4b7
http://hdl.handle.net/11386/3103826