Zobrazeno 1 - 10
of 46
pro vyhledávání: '"Klaus, Höcherl"'
Autor:
Monika Daubländer, Klaus Höcherl
Publikováno v:
Arzneiverordnungs-Report 2022 ISBN: 9783662663028
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a2b90f9605b38390da88399b6c59b3ce
https://doi.org/10.1007/978-3-662-66303-5_40
https://doi.org/10.1007/978-3-662-66303-5_40
Autor:
Klaus Höcherl, Lena R Thurner
Publikováno v:
Naunyn-Schmiedeberg's Archives of Pharmacology. 392:1401-1410
It has been reported that the serine protease kallikrein stimulates and that aprotinin, a protease inhibitor, inhibits renal renin secretion. Since direct stimulation of the protease-activated receptor (PAR) 2 increases renin secretion in isolated pe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53c34eca32f770dad0ef588ef17ed3a8
https://doi.org/10.1007/s00210-019-01677-4
https://doi.org/10.1007/s00210-019-01677-4
Autor:
Monika Daubländer, Klaus Höcherl
Publikováno v:
Arzneiverordnungs-Report 2021 ISBN: 9783662638248
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ac74db33eb986c4331d4a2183095672a
https://doi.org/10.1007/978-3-662-63825-5_45
https://doi.org/10.1007/978-3-662-63825-5_45
Autor:
Klaus Höcherl, Manuel Meurer
Publikováno v:
Pflügers Archiv - European Journal of Physiology. 471:901-914
Fibroblast growth factor 23 (FGF23) levels are elevated in patients with acute kidney injury (AKI). The consequences on renal Ca2+, Mg2+, and Pi regulatory mechanisms are unknown. We hypothesized that renal ischemia-reperfusion (I/R) injury alters th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::758f94a3589b52658cfcf9bff4c20136
https://doi.org/10.1007/s00424-019-02255-6
https://doi.org/10.1007/s00424-019-02255-6
Publikováno v:
Pflügers Archiv - European Journal of Physiology. 470:1691-1703
Endotoxemia-related acute kidney injury (AKI) is associated with increased formation of prostaglandins, which may serve as a compensatory mechanism to maintain renal function. We hypothesized that an increase of renal EP2 or EP4 receptors and/or a do
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e655d83fdaddec280801c36f6b50bee3
https://doi.org/10.1007/s00424-018-2183-3
https://doi.org/10.1007/s00424-018-2183-3
Autor:
Klaus Höcherl
Publikováno v:
BIOspektrum. 27:787-787
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::75e1f4bcdf33b66e543df76e0cd34f17
https://doi.org/10.1007/s12268-021-1655-8
https://doi.org/10.1007/s12268-021-1655-8
Autor:
Klaus Höcherl
Publikováno v:
BIOspektrum. 27:669-669
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e5a33769f315e38aa1ba074a1732eaff
https://doi.org/10.1007/s12268-021-1629-x
https://doi.org/10.1007/s12268-021-1629-x
Autor:
Bernhard M. Graf, Hendrik Busse, Michael Gruber, York Zausig, Klaus Höcherl, Timo Seyfried, Diane Bitzinger
Publikováno v:
Cardiovascular Drugs and Therapy
Introduction Mechanical and morphological ischemia and reperfusion (I/R) injury is reduced in septic hearts. The mechanism behind this “cardioprotection” is less well understood. As adenosine receptors play a major role for cardioprotection in no
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e68292cb9498dfef69fd29dd207d9e4a
https://doi.org/10.1007/s10557-016-6693-y
https://doi.org/10.1007/s10557-016-6693-y
Autor:
Manuel Meurer, Klaus Höcherl
Publikováno v:
Pflugers Archiv : European journal of physiology. 471(4)
Magnesium (Mg2+) abnormalities during sepsis have been reported, but the underlying mechanisms during acute inflammation are poorly understood. We hypothesized that a decrease in GFR and/or changes in transporters or channels for Mg2+ could be respon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4726e39e5e0cf0a4ac53db360c751a18
https://pubmed.ncbi.nlm.nih.gov/30726531
https://pubmed.ncbi.nlm.nih.gov/30726531
Publikováno v:
American Journal of Physiology-Renal Physiology. 309:F332-F340
Thromboxane (Tx) A2has been suggested to be involved in the development of sepsis-induced acute kidney injury (AKI). Therefore, we investigated the impact of cyclooxygenase (COX)-1 and COX-2 activity on lipopolysaccharide (LPS)-induced renal TxA2form
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::08ab5e8787b9323bf94512e8f381d56e
https://doi.org/10.1152/ajprenal.00567.2014
https://doi.org/10.1152/ajprenal.00567.2014