Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Kjersti D. Horvei"'
Autor:
Dhivya Thiyagarajan, Hege L. Pedersen, Natalya Seredkina, Kjersti D. Horvei, Lorena Arranz, Ramon Sonneveld, Tom Nijenhuis, Johan van der Vlag, Ole P. Rekvig
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 6 (2018)
Recently we described that endonuclease inactive DNase I translocated into the nucleus in response to increased endogenous IL-1β expression. Here, we demonstrate impact and function of translocated DNase I in tubular cells. Effect of cytokines on ex
Externí odkaz:
https://doaj.org/article/d4988ea9b4414b269dc994d85dfdd27a
Autor:
Dhivya Thiyagarajan, Hege L. Pedersen, Natalya Seredkina, Kjersti D. Horvei, Lorena Arranz, Ramon Sonneveld, Tom Nijenhuis, Johan van der Vlag, Ole P. Rekvig
Publikováno v:
Frontiers in Cell and Developmental Biology
Frontiers in Cell and Developmental Biology, 6
Frontiers in Cell and Developmental Biology, Vol 6 (2018)
Frontiers in Cell and Developmental Biology, 6
Frontiers in Cell and Developmental Biology, Vol 6 (2018)
Contains fulltext : 191059.pdf (Publisher’s version ) (Open Access) Recently we described that endonuclease inactive DNase I translocated into the nucleus in response to increased endogenous IL-1beta expression. Here, we demonstrate impact and func
Autor:
Hege L, Pedersen, Kjersti D, Horvei, Dhivya, Thiyagarajan, Gudrun E, Norby, Natalya, Seredkina, Gabriella, Moroni, Gro Ø, Eilertsen, Hallvard, Holdaas, Erik H, Strøm, Gunnstein, Bakland, Pier-Luigi, Meroni, Ole P, Rekvig
Publikováno v:
The Journal of Pathology: Clinical Research
Renal DNase I is lost in advanced stages of lupus nephritis. Here, we determined if loss of renal DNase I reflects a concurrent loss of urinary DNase I, and whether absence of urinary DNase I predicts disease progression. Mouse and human DNase I prot
Publikováno v:
The American journal of pathology. 186(11)
Divergent incommensurable models have been developed to explain the pathogenesis of lupus nephritis. Most contemporary models favor a central role for anti-chromatin antibodies. How they exert their pathogenic effect has, however, endorsed conflicts