Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Kiyokazu Nakazato"'
Autor:
Satoshi Kozawa, Ryosuke Ueda, Kyoji Urayama, Fumihiko Sagawa, Satsuki Endo, Kazuhiro Shiizaki, Hiroshi Kurosu, Glicia Maria de Almeida, Sharif M. Hasan, Kiyokazu Nakazato, Shinji Ozaki, Yoshinori Yamashita, Makoto Kuro-o, Thomas N. Sato
Publikováno v:
iScience, Vol 2, Iss , Pp 238-268 (2018)
Summary: Virtually all diseases affect multiple organs. However, our knowledge of the body-wide effects remains limited. Here, we report the body-wide transcriptome landscape across 13–23 organs of mouse models of myocardial infarction, diabetes, k
Externí odkaz:
https://doaj.org/article/40d692930b7c4ec6be5e8d98e5c5efff
Autor:
Makoto Kuro-o, Ryosuke Ueda, Glicia Maria de Almeida, Kyoji Urayama, Satsuki Endo, Satoshi Kozawa, Shinji Ozaki, Fumihiko Sagawa, Kazuhiro Shiizaki, Yoshinori Yamashita, Hiroshi Kurosu, Sharif Hasan, Thomas N. Sato, Kiyokazu Nakazato
Publikováno v:
iScience, Vol 2, Iss, Pp 238-268 (2018)
Summary: Virtually all diseases affect multiple organs. However, our knowledge of the body-wide effects remains limited. Here, we report the body-wide transcriptome landscape across 13–23 organs of mouse models of myocardial infarction, diabetes, k
Autor:
Satoshi Ogawa, Kiyokazu Nakazato, Yasuko Kitao, Hiroshi Onogi, Reiko Inagi, Kiyoshi Kurokawa, Masaomi Nangaku, William G. Couser, Hiroshi Ueyama, Toshio Miyata
Publikováno v:
Kidney International. 68:2639-2650
Involvement of endoplasmic reticulum (ER) stress in podocyte injury induced by excessive protein accumulation. Background An imbalance between protein load and folding capacity is referred to as endoplasmic reticulum (ER) stress. As a defense mechani
Autor:
Kiyoshi Kurokawa, Kiyokazu Nakazato, Yasuhiko Ueda, Toshio Miyata, Charles van Ypersele de Strihou, Satoru Takahashi, Hiroshi Onogi, Yuko Izuhara, Masayuki Yamamoto, Daisuke Suzuki, Reiko Inagi, Nobuteru Usuda, Akira Shimizu, Masaomi Nangaku, Hisashi Oishi
Publikováno v:
Journal of the American Society of Nephrology : JASN. 16(5)
The intracellular polymerization of abnormal serine protease inhibitors (serpins) results in liver or neuronal cell abnormalities recently identified as "serpinopathies." It was demonstrated in transgenic rats that overexpression of megsin, a recentl