Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kissaou Tchedre"'
Autor:
Samarendra Mohanty, Subrata Batabyal, Chinenye Idigo, Darryl Narcisse, Sanghoon Kim, Houssam Al-Saad, Michael Carlson, Kissaou Tchedre, Adnan Dibas
Publikováno v:
Journal of Translational Medicine, Vol 22, Iss 1, Pp 1-13 (2024)
Abstract Glaucoma, a blinding eye disease with optic neuropathy, is usually associated with elevated intraocular pressure (IOP). The currently available pharmacological and surgical treatments for glaucoma have significant limitations and side effect
Externí odkaz:
https://doaj.org/article/9ad284f651354ba682de5edfb1719605
Autor:
Subrata Batabyal, Sivakumar Gajjeraman, Kissaou Tchedre, Adnan Dibas, Weldon Wright, Samarendra Mohanty
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 17, Iss , Pp 758-770 (2020)
Non-viral delivery of therapeutic genes into targeted areas of retina is essential for re-functionalizing the retinal circuitry. While a focused ultrafast laser beam has been recently used for intra-ocular delivery of molecules, it poses the signific
Externí odkaz:
https://doaj.org/article/6f7c4c142a19448b972e1cc259b13a61
Autor:
Chenying Guo, Hikaru Kinouchi, Kissaou Tchedre, Dong Feng Chen, Shelley I. Fried, Seung Woo Lee, Xinghuai Sun, Yuan Fang, Kin-Sang Cho
Publikováno v:
STEM CELLS. 31:349-359
The ciliary epithelium (CE) of adult mammals has been reported to provide a source of retinal stem cells (RSCs) that can give rise to all retinal cell types in vitro. A recent study, however, suggests that CE-derived cells possess properties of pigme
Publikováno v:
Journal of Neurochemistry. 122:487-500
J. Neurochem. (2012) 122, 487–500. Abstract Genetic deletion or mutations of presenilin genes (PS1/PS2) cause familial Alzheimer’s disease and calcium (Ca2+) signaling abnormalities. PS1/PS2 act as endoplasmic reticulum (ER) Ca2+ leak channels th
Publikováno v:
Journal of neurochemistry. 122(3)
Genetic deletion or mutations of presenilin genes (PS1/PS2) cause familial Alzheimer's disease and calcium (Ca²⁺) signaling abnormalities. PS1/PS2 act as endoplasmic reticulum (ER) Ca²⁺ leak channels that facilitate passive Ca²⁺ leak across