Zobrazeno 1 - 10
of 69
pro vyhledávání: '"Kimm J. Hamann"'
Autor:
Chun-Su Yuan, Kimm J. Hamann, Jing Li, Zuo-Hui Shao, Willard W. Sharp, Terry L. Vanden Hoek, Chang Qing Li, Huiping Liu, Mei Han, Hsien Hao Haung, Srinivasan Ramachandran, Wei-Tien Chang
Publikováno v:
Journal of Cellular Biochemistry. 112:2873-2881
The cardiotoxicity of doxorubicin limits its clinical use in the treatment of a variety of malignancies. Previous studies suggest that doxorubicin-associated cardiotoxicity is mediated by reactive oxygen species (ROS)-induced apoptosis. We therefore
Autor:
Zuo-Hui Shao, Kimberly R. Wojcik, Yimin Qin, Chang-Qing Li, Terry L. Vanden Hoek, Kimm J. Hamann
Publikováno v:
Journal of Cell Death, Vol 2011, Iss 4, Pp 7-18 (2011)
Autor:
Travis Anderson, Terry L. Vanden Hoek, Kim Wojcik, Yimin Qin, Zuo-Hui Shao, Wei-Tien Chang, Kimm J. Hamann, Lance B. Becker, Juan Li, Chin-Wang Hsu, Chang Qing Li, Kim Chai Chan
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 292:H1995-H2003
Optimal timing of therapeutic hypothermia for cardiac ischemia is unknown. Our prior work suggests that ischemia with rapid reperfusion (I/R) in cardiomyocytes can be more damaging than prolonged ischemia alone. Also, these cardiomyocytes demonstrate
Autor:
Rebecca A. Shilling, Diwakar D. Balachandran, Bohao Chen, Jiankun Tong, Joel V. Weinstock, Kimm J. Hamann, Anne I. Sperling, Julian Solway, Robert A. Anders, Hozefa S. Bandulwala, Bryan S. Clay
Publikováno v:
The Journal of Experimental Medicine
Persistent airway inflammation, mucus production, and airway hyperreactivity are the major contributors to the frequency and severity of asthma. Why lung inflammation persists in asthmatics remains unclear. It has been proposed that Fas-mediated apop
Autor:
Travis Anderson, Kimm J. Hamann, Kim Wojcik, Terry L. Vanden Hoek, Zuo-Hui Shao, Yimin Qin, Lance B. Becker, Chang Qing Li
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 286:H2280-H2286
We recently demonstrated that reperfusion rapidly induces the mitochondrial pathway of apoptosis in chick cardiomyocytes after 1 h of simulated ischemia. Here we tested whether ischemia-reperfusion (I/R)-induced apoptosis could be initiated by caspas
Autor:
Franklin D. Ko, Blanca Camoretti-Mercado, Lyubov Blokh, Yimin Qin, Catherine G. Long, Kimm J. Hamann
Publikováno v:
The Journal of Immunology. 169:3536-3544
TNF family receptors can lead to the activation of NF-kappaB and this can be a prosurvival signal in some cells. Although activation of NF-kappaB by ligation of Fas (CD95/Apo-1), a member of the TNFR family, has been observed in a few studies, Fas-me
Autor:
Angelo Y. Meliton, Xiangdong Zhu, Kimm J. Hamann, Shigeharu Myou, Yimin Qin, Saori Myo, Nilda M. Munoz, Alan R. Leff, Anissa Lambertino, Evan Boetticher
Publikováno v:
Immunology. 107:77-85
We examined the role of cell surface clustering of beta2-integrin caused by protein kinase C (PKC)-activated-cPLA2 in adhesion of eosinophilic AML14.3D10 (AML) cells. Phorbol 12-myristate 13-acetate (PMA) caused time- and concentration-dependent adhe
Autor:
Lili Farrokh-Siar, Kimm J. Hamann, Jean Maguire van Seventer, Henrietta Rajadurai, Ellen M. Palmer, S. C. Patel, Kourous A. Rezai, Gijs A. van Seventer, J. Terry Ernest
Publikováno v:
Current Eye Research. 24:206-213
To evaluate whether human fetal retinal pigment epithelial (HFRPE) cells express TRAIL (tumor necrosis factor related apoptosis inducing ligand). The role of TRAIL in HFRPE induced apoptosis was evaluated.Pure cultures of HFRPE cells were isolated. T
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology. 277:L802-L810
We examined the effect of eosinophil ligation to cultured human umbilical vein endothelial cells (HUVECs) in augmenting the stimulated secretion of leukotriene (LT) C4and eosinophil peroxidase (EPO). The effects of adhesion were compared before and a
Publikováno v:
Histochemical Journal. 31:145-151
Glycosylated structures on the cell surface have a role in cell adhesion, migration, and proliferation. Repair of the airway epithelium after injury requires each of these processes, but the normal cell surface glycosylation of non-mucin producing ai