Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Kimberley Crespo"'
Autor:
Imane El. Kasmi, Amélie Bourdiec, Kimberley Crespo, Laurence Bissonnette, François Bissonnette, Soumaya Messaoudi, Cécile Le Saint, Isaac Jacques Kadoch
Publikováno v:
Fertility Research and Practice
Fertility Research and Practice, Vol 5, Iss 1, Pp 1-9 (2019)
Fertility Research and Practice, Vol 5, Iss 1, Pp 1-9 (2019)
Over the course of the last four decades, IVF has allowed an increasing number of infertile couples the chance to conceive. Considering the extensive research and advances in ART, too many IVF attempts still do not result in a successful pregnancy [1
Autor:
S. Phillips, Bernard Couturier, François Bissonnette, Sandra Bisotto, Cécile Le Saint, Isaac Jacques Kadoch, John S. Sampalis, Karen Buzaglo, Samir Hamamah, Melissa Stutz, Jean-Noël Gouze, Amélie Bourdiec, Kimberley Crespo
Publikováno v:
Reproductive BioMedicine
Reproductive BioMedicine Online, Elsevier, 2019, 38 (3), pp.321-329. ⟨10.1016/j.rbmo.2018.12.039⟩
Reproductive BioMedicine Online, Elsevier, 2019, 38 (3), pp.321-329. ⟨10.1016/j.rbmo.2018.12.039⟩
International audience; Does autologous endometrial cell co-culture (AECC) improve the number of good-quality blastocysts obtained by IVF/intracytoplasmic sperm injection (ICSI), compared with conventional embryo culture medium in a broad group of pa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5ca13914263e7ac18e7aca03282a77ca
https://doi.org/10.1016/j.rbmo.2018.12.039
https://doi.org/10.1016/j.rbmo.2018.12.039
Publikováno v:
Human Molecular Genetics. 22:4451-4459
Hypertension, the most frequently diagnosed clinical condition world-wide, predisposes individuals to morbidity and mortality, yet its underlying pathological etiologies are poorly understood. So far, a large number of quantitative trait loci (QTLs)
Autor:
Stanley Nattel, Nathalie L'Heureux, Julie Roy, Kimberley Crespo, Danielle Gelinas, Annie Ménard, Cristina Chauvet, Francine Duval, Yanfen Shi, Jean-Claude Tardif, Alan Y. Deng
Publikováno v:
Journal of Hypertension. 31:927-935
Objectives: Diastolic dysfunction often represents the onset of diastolic heart failure (DHF). We previously showed in principle that diastolic function in Dahl salt-sensitive rats (DSS) can be genetically determined by quantitative trait loci (QTLs)
Publikováno v:
Clinical and experimental hypertension (New York, N.Y. : 1993). 38(6)
Gene discovery in animal models may lead to the revelation of therapeutic targets for essential hypertension as well as mechanistic insights into blood pressure (BP) regulation. Our aim was to identify a disease-causing gene for a component of polyge
Publikováno v:
Journal of Hypertension. 29:460-465
Objectives Inbred rodent models simulating essential hypertension and normotension are useful tools in discovering genes controlling blood pressure (BP) homeostasis. An analysis of a F2 population made from crosses of hypertensive Dahl salt-sensitive
Publikováno v:
Circulation. Cardiovascular genetics. 8(4)
Background— Genetics of high blood pressure (BP) has revealed causes of hypertension. The cause of normotension, however, is poorly understood. Inbred Lewis rats sustain normotension despite a genetic push in altering BP. It was unknown whether thi
Autor:
Stanley Nattel, Nathalie L'Heureux, Danielle Gelinas, Yanfen Shi, Jean-Claude Tardif, Cristina Chauvet, Francine Duval, Alan Y. Deng, Kimberley Crespo
Publikováno v:
The Canadian journal of cardiology. 29(10)
Background Diastolic dysfunction often precedes the onset of diastolic heart failure. We previously demonstrated that diastolic dysfunction and left ventricular hypertrophy (LVH) in Dahl salt-sensitive rats can be ameliorated by quantitative trait lo
Autor:
Annie Ménard, Cristina Chauvet, Marilyne Blain, Chunjie Xiao, Julie Roy, Alan Y. Deng, Kimberley Crespo, Yanrui Wu
Publikováno v:
Journal of hypertension. 29(7)
Objectives The interval harboring a quantitative trait locus for blood pressure (BP), C18QTL3, contains β-2 adrenergic receptor (Adrb2) and neural precursor cell expressed, developmentally downregulated 4-like (Nedd4l) genes. None of the other genes