Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Kim T Vo"'
Autor:
Miguel Aste-Amézaga, Ningyan Zhang, Janet E Lineberger, Beth A Arnold, Timothy J Toner, Mingcheng Gu, Lingyi Huang, Salvatore Vitelli, Kim T Vo, Peter Haytko, Jing Zhang Zhao, Frederic Baleydier, Sarah L'Heureux, Hongfang Wang, Wendy R Gordon, Elizabeth Thoryk, Marie Blanke Andrawes, Kittichoat Tiyanont, Kimberly Stegmaier, Giovanni Roti, Kenneth N Ross, Laura L Franlin, Hui Wang, Fubao Wang, Michael Chastain, Andrew J Bett, Laurent P Audoly, Jon C Aster, Stephen C Blacklow, Hans E Huber
Publikováno v:
PLoS ONE, Vol 5, Iss 2, p e9094 (2010)
Notch receptors normally play a key role in guiding a variety of cell fate decisions during development and differentiation of metazoan organisms. On the other hand, dysregulation of Notch1 signaling is associated with many different types of cancer
Externí odkaz:
https://doaj.org/article/b4ac98e727c04ff3ad08212b68b47d08
Autor:
Yukitaka Ushio, Christopher J. Joynes, Hideyuki Saya, Isamu Okamoto, Hiromasa Tsuiki, Kim T. Vo, Andrew K. Godwin, Mitsuhiro Matsumoto, Lawrence C. Kenyon, Albert J. Wong, Irene S. Lanham, Abhijit Guha, David R. Emlet, Marina Holgado-Madruga
Publikováno v:
The American Journal of Pathology. 160:441-447
Cell surface adhesion molecules are crucial for the development and/or pathogenesis of various diseases including cancer. CD44 has received much interest as a major adhesion molecule that is involved in tumor progression. We have previously demonstra
Publikováno v:
Physiology & Behavior. 59:325-340
Flash evoked potentials (FEPs) undergo within-and between-session changes and are modified by auditory white noise (26). We examined whether an auditory potential produced by the “click” associated with the strobe discharge could be recorded, and
Autor:
Hiromasa, Tsuiki, Mehdi, Tnani, Isamu, Okamoto, Lawrence C, Kenyon, David R, Emlet, Marina, Holgado-Madruga, Irene S, Lanham, Christopher J, Joynes, Kim T, Vo, Albert J, Wong
Publikováno v:
Cancer research. 63(1)
The c-Jun NH(2)-terminal kinases (JNKs) have a role both in promoting apoptosis and tumorigenesis. The JNKs are encoded by three separate genes (JNK1, 2, and 3), which are spliced alternatively to create 10 JNK isoforms that are either M(r) 55,000 or