Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Kim E. Olson"'
Autor:
Richard R. Furman, Joan H.F. Drosopoulos, Kim E. Olson, Dianne Pulte, Aaron J. Marcus, M. Johan Broekman, Harold S. Ballard, Jorge R. Kizer
Publikováno v:
Clinical Lymphoma Myeloma and Leukemia. 11:367-372
Chronic lymphocytic leukemia (CLL) is a B-cell disorder, but it is also associated with abnormalities in T-lymphocyte function. In this study we examine changes in T-lymphocyte CD39 and CD73 expression in patients with CLL.Blood samples were drawn fr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9e027370cac1dd9bb15d2a8d1b6ad274
https://doi.org/10.1016/j.clml.2011.06.005
https://doi.org/10.1016/j.clml.2011.06.005
Autor:
Roberto Levi, Aaron J. Marcus, Joan H.F. Drosopoulos, Kim E. Olson, Naziba Islam, David J. Pinsky, M. Johan Broekman
Publikováno v:
Seminars in Thrombosis and Hemostasis. 31:234-246
Blood platelets maintain vascular integrity and promote primary and secondary hemostasis following interruption of vessel continuity. Biochemical or physical damage to coronary, carotid, or peripheral arteries promotes excessive platelet activation a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5973dff679debaeb911e15a2dd5bda59
https://doi.org/10.1055/s-2005-869528
https://doi.org/10.1055/s-2005-869528
Autor:
Ranjini Anand, Aaron J. Marcus, Alice Marino, Kengo Tomita, Silvia Aldi, Roberto Levi, Kim E. Olson, Federico Corti
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, Vol. 29, no.1, p. 61-9 (2015)
Ischemia/reperfusion (I/R) elicits renin release from cardiac mast cells (MC), thus activating a local renin-angiotensin system (RAS), culminating in ventricular fibrillation. We hypothesized that in I/R, neurogenic ATP could degranulate juxtaposed M
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d50057a009c5b1ac9154bcd2c6b80c2
https://pubmed.ncbi.nlm.nih.gov/25318477
https://pubmed.ncbi.nlm.nih.gov/25318477
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 337(2)
Once released, norepinephrine is removed from cardiac synapses via reuptake into sympathetic nerves, whereas transmitter ATP is catabolized by ecto-NTP diphosphohydrolase 1 (E-NTPDase1)/CD39, an ecto-ATPase. Because ATP is known to modulate neurotran
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::20bf229a6f0eb45a29927f6c2854e2ca
https://pubmed.ncbi.nlm.nih.gov/21325440
https://pubmed.ncbi.nlm.nih.gov/21325440
Autor:
J.K. Chan, Kim E. Olson, C.W. Olson, Ronald H. Selvester, D.M. Lange, Galen S. Wagner, G.D. Bass
Publikováno v:
2007 Computers in Cardiology.
The objective of this paper is to develop a model for simulation of bundle branch and fascicular block. The model accounts for the contribution of fiber orientation to the velocity of signal propagation through the myocardium as well as "reentry " of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0e098adb0ad7bb5b2b47aacc2083ddbb
https://doi.org/10.1109/cic.2007.4745489
https://doi.org/10.1109/cic.2007.4745489
Autor:
Kim E. Olson, Jorge R. Kizer, Aaron J. Marcus, Joan H.F. Drosopoulos, E. Dianne Pulte, M. Johan Broekman, Naziba Islam
Publikováno v:
Thrombosis research. 121(3)
Introduction CD39/NTPDase-1 is a cell surface enzyme expressed on leukocytes and endothelial cells that metabolizes ATP to ADP and AMP. CD39 is expressed on numerous different types of normal leukocytes, but details of its expression have not been de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f9eb2097ae9c681c0bc2ea8a03ab4f73
https://pubmed.ncbi.nlm.nih.gov/17555802
https://pubmed.ncbi.nlm.nih.gov/17555802
Autor:
Kim E. Olson, Alfred Albano, Charles W. Olson, David Lange, Jack-Kang Chan, Ronald H. Selvester, Galen S. Wagner
Publikováno v:
Journal of electrocardiology. 40(5)
This new training method is based on developing a sound understanding of the sequence in which electrical excitation spreads through both the normal and the infarcted myocardium. The student is made aware of the cardiac electrical performance through
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b83b7815957aa956678e362b3786f3e4
https://pubmed.ncbi.nlm.nih.gov/17604044
https://pubmed.ncbi.nlm.nih.gov/17604044
Autor:
Yasushi Yoshikawa, Shi Fang Yan, Catherine Wang, Kim E. Olson, Morihito Okada, Sean P. Mazer, David J. Pinsky, Taichi Sakaguchi, Kanji Minamoto, Daniel W. Hwang
Publikováno v:
Circulation research. 91(2)
Expression of the zinc finger transcription factor early growth response gene-1 (Egr-1) is triggered rapidly after mechanical vascular injury or after a precipitous drop in ambient oxygen, whereupon it induces the expression of diverse gene families
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79b2107f8d8edaa411342eb26986f002
https://pubmed.ncbi.nlm.nih.gov/12142346
https://pubmed.ncbi.nlm.nih.gov/12142346
Autor:
Shi Fang Yan, David M. Stern, Taichi Sakaguchi, Tucker Collins, Tomoyuki Fujita, Morihito Okada, David J. Pinsky, Kim E. Olson
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 15(14)
Hypoxic induction of the early growth response-1 (Egr-1) transcription factor initiates proinflammatory and procoagulant gene expression. Orthotopic/isogeneic rat lung transplantation triggers Egr-1 expression and nuclear DNA binding activity corresp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8563682c55b2a283b5c3c15c259cefa0
https://pubmed.ncbi.nlm.nih.gov/11606484
https://pubmed.ncbi.nlm.nih.gov/11606484
Nitric oxide triggers programmed cell death (apoptosis) of adult rat ventricular myocytes in culture
Autor:
Kim E. Olson, Youping Liu, Yi Ming Yang, David J. Pinsky, Richard P. Kline, Matthias Szabolcs, Paul J. Cannon, Eleni Athan, Walif Aji
Publikováno v:
The American journal of physiology. 277(3)
Excessive nitric oxide (NO) production within the heart is implicated in the pathogenesis of myocyte death, but the mechanism whereby NO kills cardiac myocytes is not known. To determine whether NO may trigger programmed cell death (apoptosis) of adu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ab7bb5a33f47a9560aa1bfdd157ce9d
https://pubmed.ncbi.nlm.nih.gov/10484441
https://pubmed.ncbi.nlm.nih.gov/10484441