Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Khosbayar Lkhagvadorj"'
Autor:
Khosbayar Lkhagvadorj, Karolin F. Meyer, Laura P. Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Henk W. Dijkhuizen, Inge A. M. de Graaf, Torsten Plösch, Machteld N. Hylkema
Publikováno v:
Epigenetics, Vol 15, Iss 12, Pp 1370-1385 (2020)
Prenatal smoke exposure (PSE) is a risk factor for nicotine dependence. One susceptibility gene for nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conversion of nicotine to cotinine and nicotine clearance in the liver
Externí odkaz:
https://doaj.org/article/9c83266db7144cbbba7fce50a2335ecf
Autor:
Khosbayar Lkhagvadorj, Zhijun Zeng, Karolin F. Meyer, Laura P. Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Henk W. Dijkhuizen, Inge A. M. de Graaf, Torsten Plösch, Machteld N. Hylkema
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 1, p 164 (2020)
Prenatal smoke exposure (PreSE) is a risk factor for nicotine dependence, which is further enhanced by postnatal smoke exposure (PostSE). One susceptibility gene to nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conve
Externí odkaz:
https://doaj.org/article/7ce5be2cb7c74864942a998f51e3c67a
Autor:
Karolin F Meyer, Henk W Dijkhuizen, Torsten Plösch, Laura P Verweij, Wierd Kooistra, Marjan Reinders-Luinge, Khosbayar Lkhagvadorj, Machteld N. Hylkema, Inge A. M. de Graaf
Publikováno v:
Epigenetics, 15(12), 1370-1385. TAYLOR & FRANCIS INC
Prenatal smoke exposure (PSE) is a risk factor for nicotine dependence. One susceptibility gene for nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conversion of nicotine to cotinine and nicotine clearance in the liver
Autor:
Machteld N. Hylkema, Wierd Kooistra, Marjan Reinders-Luinge, Juan Song, Torsten Plösch, Xia Huo, Zhijun Zeng, Karolin F Meyer, Khosbayar Lkhagvadorj, Xijin Xu
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology, 318(3), L549-L561. AMER PHYSIOLOGICAL SOC
Prenatal smoke exposure (PSE) is associated with reduced birth weight, impaired fetal development, and increased risk for diseases later in life. Changes in DNA methylation may be involved, as multiple large-scale epigenome-wide association studies s
Autor:
Khosbayar Lkhagvadorj
Lung progenitor cell behavior and Cyp2a5 gene regulationPrenatal smoke exposure (PSE) is associated with an increased risk for chronic obstructive pulmonary disease (COPD) and cigarette smoke addiction in later in life. One of the proposed underlying
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9fa52347d6926a833d0bc60012740b08
https://doi.org/10.33612/diss.171373691
https://doi.org/10.33612/diss.171373691
Autor:
Machteld N. Hylkema, Henk W Dijkhuizen, Torsten Plösch, Wierd Kooistra, Zhijun Zeng, Karolin F Meyer, Laura P Verweij, Khosbayar Lkhagvadorj, Inge A. M. de Graaf, Marjan Reinders-Luinge
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 164, p 164 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 1
International Journal of Molecular Sciences, 22(1):164. MDPI AG
International Journal of Molecular Sciences
Volume 22
Issue 1
International Journal of Molecular Sciences, 22(1):164. MDPI AG
Prenatal smoke exposure (PreSE) is a risk factor for nicotine dependence, which is further enhanced by postnatal smoke exposure (PostSE). One susceptibility gene to nicotine dependence is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conve
Autor:
Juan Song, Khosbayar Lkhagvadorj, Barbro N. Melgert, Susanne Krauss-Etschmann, Shanshan Song, Wierd Kooistra, Zhijun Zeng, Marjan Reinders-Luinge, Machteld N. Hylkema, Junjun Cao
Publikováno v:
American Journal of Physiology-Lung Cellular and Molecular Physiology, 319(4), L742-L751. AMER PHYSIOLOGICAL SOC
Prenatal smoke exposure is a risk factor for impaired lung development in children. Recent studies have indicated that amphiregulin (AREG), which is a ligand of the epidermal growth factor receptor (EGFR), has a regulatory role in airway epithelial c
Autor:
Zhijun, Zeng, Karolin F, Meyer, Khosbayar, Lkhagvadorj, Wierd, Kooistra, Marjan, Reinders-Luinge, Xijin, Xu, Xia, Huo, Juan, Song, Torsten, Plösch, Machteld N, Hylkema
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 318(3)
Prenatal smoke exposure (PSE) is associated with reduced birth weight, impaired fetal development, and increased risk for diseases later in life. Changes in DNA methylation may be involved, as multiple large-scale epigenome-wide association studies s
Autor:
Laura P Verweij, Torsten Plösch, Khosbayar Lkhagvadorj, Karolin F Meyer, Zhijun Zeng, Machteld N. Hylkema
Publikováno v:
European Respiratory Journal, 52(Suppl.62). EUROPEAN RESPIRATORY SOC JOURNALS LTD
Background: Prenatal smoke exposure (PSE) is a risk factor for COPD. One susceptibility gene for COPD is Cytochrome P450 (CYP) 2A6, an enzyme responsible for the conversion of nicotine to cotinine and nicotine clearance in the liver. Higher CYP2A6 ac