Zobrazeno 1 - 10
of 51
pro vyhledávání: '"Kevin K. Frick"'
Autor:
Felix M. Ramos, Kevin K. Frick, Amy VandenEynde, Nancy Krieger, Ignacio Granja, David A. Bushinsky, Min Ho Kim, John R. Asplin, Marc D. Grynpas
Publikováno v:
The Journal of urology. 202(4)
Background: Urine (u) calcium (Ca) excretion is directly dependent on dietary sodium (Na) intake leading to the recommendation for Na restriction in hypercalciuric kidney stone formers. However, there is no direct evidence that limiting Na intake wil
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9f91a8a34b7bf7a561b5238755d34d1c
https://pubmed.ncbi.nlm.nih.gov/30726853
https://pubmed.ncbi.nlm.nih.gov/30726853
Autor:
Ignacio Granja, Christopher D. Culbertson, David A. Bushinsky, Marc D. Grynpas, John R. Asplin, Nancy Krieger, Kevin K. Frick, Adeline Ng
Publikováno v:
Journal of the American Society of Nephrology. 26:3001-3008
Potassium citrate is prescribed to decrease stone recurrence in patients with calcium nephrolithiasis. Citrate binds intestinal and urine calcium and increases urine pH. Citrate, metabolized to bicarbonate, should decrease calcium excretion by reduci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4cc5f4c6b8e8e43ec4badcb23b6cb47
https://doi.org/10.1681/asn.2014121223
https://doi.org/10.1681/asn.2014121223
Autor:
Kevin K. Frick, Christopher D. Culbertson, Adeline H. Ng, Madison Cohen-McFarlane, Marc D. Grynpas, Nancy Krieger, John R. Asplin, David A. Bushinsky, Kelly Kyker-Snowman
Publikováno v:
Calcified Tissue International. 94:531-543
Genetic hypercalciuric stone-forming (GHS) rats, bred to maximize urine (u) calcium (Ca) excretion, demonstrate increased intestinal Ca absorption, increased bone Ca resorption, and reduced renal Ca reabsorption, all leading to elevated uCa compared
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1ef4f53150222958f9f60f807dbe1dce
https://doi.org/10.1007/s00223-014-9838-7
https://doi.org/10.1007/s00223-014-9838-7
Autor:
David A. Bushinsky, John R. Asplin, Nancy Krieger, Kevin K. Frick, Daniel M. Asplin, Christopher D. Culbertson
Publikováno v:
American Journal of Physiology-Renal Physiology. 305:F1132-F1138
The inbred genetic hypercalciuric stone-forming (GHS) rats exhibit many features of human idiopathic hypercalciuria and have elevated levels of vitamin D receptors (VDR) in calcium (Ca)-transporting organs. On a normal-Ca diet, 1,25(OH)2D3 (1,25D) in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8aa46d332a4e41944dac80c6abb2433d
https://doi.org/10.1152/ajprenal.00296.2013
https://doi.org/10.1152/ajprenal.00296.2013
Publikováno v:
Journal of Bone and Mineral Research. 22:907-917
Chronic metabolic acidosis induces net Ca efflux from bone; this osteoclastic bone resorption is mediated by increased osteoblastic prostaglandin synthesis. Cyclooxygenase, the rate-limiting enzyme in prostaglandin synthesis, is present in both const
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e367d8ac3f7358ee5173f3603b0f294f
https://doi.org/10.1359/jbmr.070316
https://doi.org/10.1359/jbmr.070316
Publikováno v:
Current opinion in nephrology and hypertension. 24(4)
In this review, we discuss how the genetic hypercalciuric stone-forming (GHS) rats, which closely model idiopathic hypercalciuria and stone formation in humans, provide insights into the pathophysiology and consequences of clinical hypercalciuria. Hy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ac0df9d12bbb0dcc37ec01849e0927dd
https://pubmed.ncbi.nlm.nih.gov/26050120
https://pubmed.ncbi.nlm.nih.gov/26050120
Publikováno v:
American Journal of Physiology-Renal Physiology. 289:F1005-F1011
Chronic metabolic acidosis stimulates net calcium efflux from bone due to increased osteoclastic bone resorption and decreased osteoblastic collagen synthesis. Previously, we determined that incubation of neonatal mouse calvariae in medium simulating
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37c3d5142dea2c45a470e9948da3b224
https://doi.org/10.1152/ajprenal.00420.2004
https://doi.org/10.1152/ajprenal.00420.2004
Publikováno v:
Seminars in Dialysis. 16:463-466
Metabolic acidosis increases urine calcium excretion without an increase in intestinal calcium absorption, resulting in a net loss of bone mineral. In vitro metabolic acidosis induces bone calcium efflux initially by physicochemical dissolution and s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1bdda59cb3c43ab54fa91986efa825ec
https://doi.org/10.1046/j.1525-139x.2003.16100.x
https://doi.org/10.1046/j.1525-139x.2003.16100.x
Publikováno v:
Journal of the American Society of Nephrology. 13:2534-2539
Metabolic acidosis increases urine calcium excretion without an increase in intestinal calcium absorption, resulting in a net loss of bone mineral. In vitro, metabolic acidosis has been shown to initially induce physicochemical mineral dissolution an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d13de23a4d29a71033a5c86d0e2c8f9f
https://doi.org/10.1097/01.asn.0000031721.19801.7c
https://doi.org/10.1097/01.asn.0000031721.19801.7c
Autor:
Nancy Krieger, Ignacio Granja, John R. Asplin, Christopher D. Culbertson, David A. Bushinsky, Kevin K. Frick
Publikováno v:
American journal of physiology. Renal physiology. 306(9)
Genetic hypercalciuric stone-forming (GHS) rats demonstrate increased intestinal Ca absorption, increased bone resorption, and reduced renal tubular Ca reabsorption leading to hypercalciuria and all form kidney stones. GHS have increased vitamin D re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd52094dd035ce77317e2b9ac73bff45
https://pubmed.ncbi.nlm.nih.gov/25035042
https://pubmed.ncbi.nlm.nih.gov/25035042