Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Kerrie Way"'
Autor:
John A. Hamilton, Andrew D. Cook, Felix I.L. Clanchy, Kirby E White, A. Ian Cassady, Martin R Keene, Patricia E Lusby, John Roiniotis, Hang Dinh, David J. Curtis, Kerrie Way
Publikováno v:
Journal of leukocyte biology. 85(5)
Information about the development and function of human macrophage lineage populations, such as osteoclasts, is limited because of the lack of defined in vitro systems for their large-scale generation. Two M-CSF-containing cytokine cocktails were fou
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ff2e5d094f4e3afe18855d621968a47c
https://pubmed.ncbi.nlm.nih.gov/19181863
https://pubmed.ncbi.nlm.nih.gov/19181863
Autor:
Piotr Witkowski, Kerrie Way, Guosheng Xiang, Michael D. Schuster, Andrew J. Boyle, Silviu Itescu, Tetsunori Seki
Publikováno v:
Journal of the Renin-Angiotensin-Aldosterone System, Vol 6 (2005)
Animal studies have demonstrated the efficacy of endothelial progenitor cells (EPCs) in preventing left ventricular (LV) remodelling following myocardial infarction (MI). Preliminary human studies are underway, yet no studies have demonstrated effica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d260e4a0f8b7b535afd3568a60d3755
https://pubmed.ncbi.nlm.nih.gov/16088849
https://pubmed.ncbi.nlm.nih.gov/16088849
Autor:
Yuan Zhang, Darren J. Kelly, Andrew J. Boyle, Richard E. Gilbert, Silviu Itescu, Renae M. Gow, Kerrie Way, Henry Krum, Alison J. Cox
Publikováno v:
Journal of molecular and cellular cardiology. 39(2)
Despite current therapies, chronic heart failure (CHF) remains a major complication of myocardial infarction (MI). The pathological changes that follow MI extend to regions remote from the site of infarction (non-infarct zone, NIZ) where fibrosis is
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e3f5f80ce80b160aa99c8c0449387a9
https://pubmed.ncbi.nlm.nih.gov/15878171
https://pubmed.ncbi.nlm.nih.gov/15878171
Autor:
Henry Krum, Andrew R Kompa, Dion Lewis, Silviu Itescu, Kerrie Way, Fiona See, Alex Tzanidis, Walter G. Thomas
Publikováno v:
Journal of the American College of Cardiology. 44(8)
OBJECTIVES: The aim of this study was to examine the effect of the p38 mitogen-activated protein kinase (MAPK) inhibitor, RWJ-67657 (RWJ), on left ventricular (LV) dysfunction and remodeling post-myocardial infarction (MI) in rats. BACKGROUND: p38 MA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63ef77a57b73409a2c6a4be16c4d55eb
https://pubmed.ncbi.nlm.nih.gov/15489104
https://pubmed.ncbi.nlm.nih.gov/15489104