Výsledky vyhledávání - "Keren B. Turton"

Akademický článek

Autor: Deane F. Mosher, Emily M. Wilkerson, Keren B. Turton, Alexander S. Hebert, Joshua J. Coon

Publikováno v: Frontiers in Medicine, Vol 4 (2017)

We recently identified and quantified >7,000 proteins in non-activated human peripheral blood eosinophils using liquid chromatography coupled to tandem mass spectrometry (LC–MS/MS) and described phosphoproteomic changes that accompany acute activat

Externí odkaz: https://doaj.org/article/dc33877f22f84ea8bffe19aca4700671

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Macrophage dysfunction in cystic fibrosis: Nature or nurture?

Autor: Rebecca J. Ingram, Miguel A. Valvano, Keren B. Turton

Publikováno v: Turton, K, Ingram, R J & Valvano, M A 2020, ' Macrophage dysfunction in cystic fibrosis: nature or nurture? ', Journal of Leukocyte Biology . https://doi.org/10.1002/JLB.4RU0620-245R

Mutations in the cystic fibrosis transmembrane conductance regulator (CFTR) affect the home-ostasis of chloride flux by epithelial cells. This has deleterious consequences, especially in respira-tory epithelia, where the defect results in mucus accum

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::81d4218f30a6600d39e1e7ff0543d268
https://doi.org/10.1002/jlb.4ru0620-245r

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Myeloid-derived growth factor is a resident endoplasmic reticulum protein

Autor: Karina T. Barretto, Frances J. Fogerty, Douglas S. Annis, Valeriu Bortnov, Keren B. Turton, Deane F. Mosher

Publikováno v: Journal of Biological Chemistry. 293:13166-13175

Human myeloid-derived growth factor (MYDGF; also known as C19orf10) is named based on its identification as a secreted monocyte/macrophage–derived mediator of cardiac repair following myocardial infarction in mice. Homologs of MYDGF, however, are p

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1465fcc299c4ccef17c5d8ff45cf189c
https://doi.org/10.1074/jbc.ac118.002052

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Expression of novel 'LOCGEF' isoforms of ARHGEF18 in eosinophils

Autor: Keren B. Turton, Emily M. Wilkerson, Hazel M. Schira, Fady E. Botros, Joshua J. Coon, Deane F. Mosher, Alexander S. Hebert, Frances J. Fogerty

Publikováno v: Journal of leukocyte biology. 104(1)

Genomic, transcriptomic and proteomic databases indicate that the N-terminal 322 residues encoded by the presumptive LOC100996504 gene, which is adjacent to the ARHGEF18 guanine nucleotide exchange factor gene on chromosome 19, constitute the N-termi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::52944e6d28ca1e6d14cb143c2963d2cc
https://pubmed.ncbi.nlm.nih.gov/29601110

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Merging Absolute and Relative Quantitative PCR Data to Quantify STAT3 Splice Variant Transcripts

Autor: Larissa P. Delain, Stephane Esnault, Keren B. Turton, Deane F. Mosher

Publikováno v: Turton, K, Esnault, S, Delain, L & Mosher, D 2016, ' Merging Absolute and Relative Quantitative PCR Data to Quantify STAT3 Splice Variant Transcripts. ', Journal of visualized experiments : JoVE, vol. 116 . https://doi.org/10.3791/54473
Journal of Visualized Experiments : JoVE

Human signal transducer and activator of transcription 3 (STAT3) is one of many genes containing a tandem splicing site. Alternative donor splice sites 3 nucleotides apart result in either the inclusion (S) or exclusion (ΔS) of a single residue, Ser

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7a040691396dc32d6f74bc30416311e1
https://doi.org/10.3791/54473

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Ratios of Four STAT3 Splice Variants in Human Eosinophils and Diffuse Large B Cell Lymphoma Cells

Autor: Keren B. Turton, Douglas S. Annis, Stephane Esnault, Deane F. Mosher, Lixin Rui

Publikováno v: PLoS ONE, Vol 10, Iss 5, p e0127243 (2015)
PLoS ONE

Signal transducer and activator of transcription 3 (STAT3) is a key mediator of leukocyte differentiation and proliferation. The 3' end of STAT3 transcripts is subject to two alternative splicing events. One results in either full-length STAT3α or i

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::737ffc50c07aaa03e9f6bc71b33f64f5
http://europepmc.org/articles/PMC4436176?pdf=render

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A mix of S and ΔS variants of STAT3 enable survival of activated B-cell-like diffuse large B-cell lymphoma cells in culture

Autor: Deane F. Mosher, Douglas S. Annis, M Zheng, Amanda C. Drennan, Fen Zhu, Li Lu, D J Tweardy, Lixin Rui, Yangguang Li, Kreg M. Grindle, U Bharadwaj, Keren B. Turton

Publikováno v: Oncogenesis

Activated B-cell-like diffuse large B-cell lymphoma (ABC DLBCL) is characterized by increased expression and activator of signal transducer and activator of transcription 3 (STAT3). ABC DLBCL cells require STAT3 for growth in culture. In ABC DLBCL ce

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b75076461e3f3932e72dcafb7f715f6c
https://doi.org/10.1038/oncsis.2015.44

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Inventory of STAT3 Splice Variants in ABC- and GCB-DLBCL

Autor: Douglas S. Annis, Deane F. Mosher, Keren B. Turton, Lixin Rui, Stephane Esnault

Publikováno v: Blood. 124:5180-5180

Signal transducer and activator of transcription 3 (STAT3) is a key mediator of lymphocyte differentiation and proliferation. Germline mutations of STAT3 are associated with hyper-IgE syndrome, recurring STAT3 somatic mutations are associated with so

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::08c26b04c26021e8cde597f5201795ba
https://doi.org/10.1182/blood.v124.21.5180.5180

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