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Autor:
Sha Chen, Kenneth Overberg, Zakiya Ghouse, Markus W. Hollmann, Nina C. Weber, Ruben Coronel, Coert J. Zuurbier
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 174, Iss , Pp 116477- (2024)
Background: SGLT2i reduce cardiac hypertrophy, but underlying mechanisms remain unknown. Here we explore a role for serine/threonine kinases (STK) and sodium hydrogen exchanger 1(NHE1) activities in SGLT2i effects on cardiac hypertrophy. Methods: Iso
Externí odkaz:
https://doaj.org/article/66c45ad5380a457c848948bdb50a2688