Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Kenian Liu"'
Publikováno v:
Leukemia Research Reports, Vol 14, Iss , Pp 100222- (2020)
We presented a patient with CML who progressed to unclassical blast phase after Tyrosine Kinase Inhibitors (TKIs) therapy. The patient presented with 2 populations of blasts: one with no cytoplasmic granules and was CD117 weak+/tryptase-/CD34- (typic
Externí odkaz:
https://doaj.org/article/598d338b4e7a48aa83dda47ae26bc7b1
Autor:
Douglas C Wilson, Gijsbert M Grotenbreg, Kenian Liu, Yanlin Zhao, Eva-Maria Frickel, Marc-Jan Gubbels, Hidde L Ploegh, George S Yap
Publikováno v:
PLoS Pathogens, Vol 6, Iss 3, p e1000815 (2010)
Production of the pro-inflammatory cytokine IL-12 by innate phagocytes drives the differentiation of IFN-gamma-producing effector T cells during Toxoplasma gondii infection. However, the role of IL-12 in the regulation of memory CD8+ T cell different
Externí odkaz:
https://doaj.org/article/4534af99bef443e29a5a6712821d0d04
Publikováno v:
Journal of the Association of Genetic Technologists. 48(4)
Fluorescence in situ hybridization (FISH) is a quick and reliable test to detect the reciprocal t(15;17)(q22;q21) translocation in acute promyeloid leukemia (APL). The typical signal pattern for positive t(15;17) is one red, one green, and two fusion
Publikováno v:
Leukemia Research Reports
Leukemia Research Reports, Vol 14, Iss, Pp 100222-(2020)
Leukemia Research Reports, Vol 14, Iss, Pp 100222-(2020)
We presented a patient with CML who progressed to unclassical blast phase after Tyrosine Kinase Inhibitors (TKIs) therapy. The patient presented with 2 populations of blasts: one with no cytoplasmic granules and was CD117 weak+/tryptase-/CD34- (typic
Publikováno v:
Advances in Modern Oncology Research, Vol 3, Iss 1, Pp 12-14 (2017)
Acute myeloid leukemia (AML) with CBFB-MYH11 fusion gene is almost always associated with either inv(16)(p13.1q22) or t(16;16)(p13.1;q22). Only a few variants leading to formation of this chimeric gene have been reported. In this report, we describe
Autor:
Lynn C. Moscinski, Huijuan Jiang, Fengdong Cheng, Nathanael S. Gray, Matthew A. Lawlor, Paul M.C. Park, John M. Koomen, Jianguo Tao, William S. Dalton, Bijal D. Shah, Michelle Wang, John L. Cleveland, Chengfeng Bi, Jing Gao, Xuefeng Wang, Kaplan Lee, Kenneth H. Shain, Tint Lwin, Mousheng Xu, Kai Fu, Kenian Liu, Tao Li, Huitao Fan, Yuan Ren, Praneeth Reddy Sudalagunta, Jun Qi, Xiaohong Zhao, Gang Greg Wang, Eduardo M. Sotomayor, Tinghu Zhang, Ariosto S. Silva, Mark B. Meads
Publikováno v:
Cancer Cell. 35:752-766.e9
Summary Drug-tolerant "persister" tumor cells underlie emergence of drug-resistant clones and contribute to relapse and disease progression. Here we report that resistance to the BCL-2 targeting drug ABT-199 in models of mantle cell lymphoma and doub
Autor:
Vander Velde, Robert, Yoon, Nara, Marusyk, Viktoriya, Durmaz, Arda, Dhawan, Andrew, Miroshnychenko, Daria, Lozano-Peral, Diego, Desai, Bina, Balynska, Olena, Poleszhuk, Jan, Kenian, Liu, Teng, Mingxiang, Abazeed, Mohamed, Mian, Omar, Tan, Aik Choon, Haura, Eric, Scott, Jacob, Marusyk, Andriy
Publikováno v:
Nature Communications; 5/14/2020, Vol. 11 Issue 1, p1-13, 13p
Autor:
F. Joseph Daugherty, Ji-Hyun Lee, Rami S. Komrokji, Alan F. List, Ling Zhang, Najla Al Ali, Gisela Caceres, William J. Fulp, Sheng Wei, Richard A. Wells, Neil Littleton, Kathy L. McGraw, Joseph O. Johnson, Jacqueline Boultwood, Andrea Pellagatti, Lubomir Sokol, Larry J. Smith, Kenian Liu, Ashley A. Basiorka, Lan Min Zhang, Bon Ham Yip
Stabilization of p53 in erythroid precursors in response to nucleosomal stress underlies the hypoplastic anemia in myelodysplastic syndromes (MDS) with chromosome 5q deletion [del(5q)]. We investigated whether cenersen, a clinically active 20-mer ant
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8c4668418894685372253ee7e83332f
https://ora.ox.ac.uk/objects/uuid:246ea8a8-470f-4543-9b1a-da60e1dfea0b
https://ora.ox.ac.uk/objects/uuid:246ea8a8-470f-4543-9b1a-da60e1dfea0b
Publikováno v:
Cytokine. 31:251-257
The estrogen receptor (ER) suppresses interleukin-6 (IL-6) gene expression through interaction with nuclear factor kappaB (NF-kappaB) in a hormone-dependent manner. Classic ER binding to DNA is not required and the mechanism of repression is unclear.
Publikováno v:
Molecular and Cellular Endocrinology. 194:51-61
In response to hormonal stimulation quiescent 3T3-L1 preadipocyte cells reenter the cell cycle and undergo a mitotic expansion phase prior to terminal differentiation. The cell cycle regulatory proteins p130 and p107 undergo dramatic changes in prote