Zobrazeno 1 - 10
of 80
pro vyhledávání: '"Ken-ichi Katsube"'
Autor:
Rumana Khanom, Chi Thi Kim Nguyen, Kou Kayamori, Xin Zhao, Keiichi Morita, Yoshio Miki, Ken-Ichi Katsube, Akira Yamaguchi, Kei Sakamoto
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0161163 (2016)
Keratin subtypes are selectively expressed depending on the cell type. They not only provide structural support, but regulate the metabolic processes and signaling pathways that control the growth of the epithelium. KRT17 (keratin 17) is induced in t
Externí odkaz:
https://doaj.org/article/52c3f4e89a504bfda1755a71c0b0d86e
Autor:
Takashi Watanabe, Toshinao Oyama, Maki Asada, Daisuke Harada, Yoshiaki Ito, Masayo Inagawa, Yutaka Suzuki, Sumio Sugano, Ken-ichi Katsube, Gerard Karsenty, Toshihisa Komori, Motoo Kitagawa, Hiroshi Asahara
Publikováno v:
PLoS Genetics, Vol 9, Iss 1, p e1003132 (2013)
Mastermind-like 1 (MAML1) is a transcriptional co-activator in the Notch signaling pathway. Recently, however, several reports revealed novel and unique roles for MAML1 that are independent of the Notch signaling pathway. We found that MAML1 enhances
Externí odkaz:
https://doaj.org/article/06f42c6c900e4bfa93410d979e79e23e
Autor:
Yasuyuki Shimada, Ken-ichi Katsube, Yuji Kabasawa, Kei-ichi Morita, Ken Omura, Akira Yamaguchi, Kei Sakamoto
Publikováno v:
PLoS ONE, Vol 8, Iss 8, p e70995 (2013)
Keratocystic odontogenic tumor (KCOT) arises as part of Gorlin syndrome (GS) or as a sporadic lesion. Gene mutations and loss of heterozygosity (LOH) of the hedgehog receptor PTCH1 plays an essential role in the pathogenesis of KCOT. However, some KC
Externí odkaz:
https://doaj.org/article/d234d6e0f9254c99932b7ad35dfd3052
Autor:
Hideyuki Tanabe, Issei Takayama, Takashi Nishiyama, Masashi Shimazaki, Isao Kii, Minqi Li, Norio Amizuka, Ken-ichi Katsube, Akira Kudo
Publikováno v:
PLoS ONE, Vol 5, Iss 8, p e12234 (2010)
BACKGROUND: Matricellular proteins, including periostin, modulate cell-matrix interactions and cell functions by acting outside of cells. METHODS AND FINDINGS: In this study, however, we reported that periostin physically associates with the Notch1 p
Externí odkaz:
https://doaj.org/article/8db963c47d0a4a849b9f8f67cb7a3cff
Autor:
Takashi Okiji, Hiroki Murano, Mitsuhiro Sunakawa, Yamato Okada, Tomoatsu Kaneko, Ken-ichi Katsube, Su Yee Myo Zaw, Phyo Pyai Sone, Zar Chi Thein Zaw
Publikováno v:
Oral Diseases. 28:1674-1681
Objectives Signals from inflamed tooth pulp activate thalamic neurons to evoke central sensitization. We aimed to gain insights into the mechanisms mediating the early phase of pulpal inflammation-induced thalamic neural and glial activation. Materia
Autor:
Takashi Okiji, Yukikio Sueyama, Peifeng Han, Su Yee Myo Zaw, Zar Chi Thein Zaw, Tomoatsu Kaneko, Hiroki Murano, Yamato Okada, Ken-ichi Katsube, Bin Gu, Phyo Pyai Sone
Publikováno v:
Journal of Endodontics. 46:943-949
Introduction This study aimed to examine the process of reinnervation during coronal pulp tissue regeneration in a rat model in which rat bone marrow mesenchymal stem cells were implanted in pulpotomized molars. Methods The maxillary first molars of
Publikováno v:
Biochemical and Biophysical Research Communications. 518:233-238
Fgf23 acts as a phosphaturic factor secreted from osteocytes in bone, but the mechanism regulating Fgf23 is not fully understood. Here, we showed the colocalization of Fgf23, Notch, and Hes1, a downstream target of Notch signaling, in numerous osteoc
Autor:
Hiroki, Murano, Tomoatsu, Kaneko, Su Yee Myo, Zaw, Phyo Pyai, Sone, Zar Chi Thein, Zaw, Yamato, Okada, Mitsuhiro, Sunakawa, Ken-Ichi, Katsube, Takashi, Okiji
Publikováno v:
Oral diseasesREFERENCES. 28(6)
Signals from inflamed tooth pulp activate thalamic neurons to evoke central sensitization. We aimed to gain insights into the mechanisms mediating the early phase of pulpal inflammation-induced thalamic neural and glial activation.Pulpal inflammation
Autor:
Tomoatsu Kaneko, Yamato Okada, Takashi Okiji, Su Yee Myo Zaw, Zar Chi Thein Zaw, Phyo Pyai Sone, Hiroki Murano, Ken-ichi Katsube, Mitsuhiro Sunakawa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a69c9c461f3982e6a9d15d4acd990a23
https://doi.org/10.1111/odi.13866/v2/response1
https://doi.org/10.1111/odi.13866/v2/response1
Crosstalk between dental pulp stem cells and endothelial cells augments angiogenic factor expression
Autor:
Takashi Okiji, Peifeng Han, Phyo Pyai Sone, Su Yee Myo Zaw, Tomoatsu Kaneko, Yamato Okada, Zar Chi Thein Zaw, Ken-ichi Katsube, Bin Gu, Hiroki Murano
Publikováno v:
Oral diseasesREFERENCES.
Objectives We aimed to investigate whether the mesenchymal stem cell-endothelial cell crosstalk enhances angiogenic factor expression via nuclear factor-kappa B (NF-κB)-dependent mechanisms. Materials and methods Human dermal microvascular endotheli