Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Keizo Gamo"'
Autor:
Keiko Nakashima, Yukie Hirahara, Taro Koike, Susumu Tanaka, Keizo Gamo, Souichi Oe, Shinichi Hayashi, Ryohei Seki-Omura, Yousuke Nakano, Chisato Ohe, Takashi Yoshida, Yosky Kataoka, Masayuki Tsuda, Tatsuyuki Yamashita, Koichi Honke, Masaaki Kitada
Publikováno v:
Journal of Lipid Research, Vol 63, Iss 6, Pp 100210- (2022)
Diverse molecular species of sulfatide with differences in FA lengths, unsaturation degrees, and hydroxylation statuses are expressed in the kidneys. However, the physiological functions of specific sulfatide species in the kidneys are unclear. Here,
Externí odkaz:
https://doaj.org/article/393b0dbd1bff4a05bc96143ea39a5f13
Autor:
Keiko Nakashima, Yukie Hirahara, Taro Koike, Susumu Tanaka, Keizo Gamo, Souichi Oe, Shinichi Hayashi, Ryohei Seki-Omura, Yousuke Nakano, Chisato Ohe, Takashi Yoshida, Yosky Kataoka, Masayuki Tsuda, Tatsuyuki Yamashita, Koichi Honke, Masaaki Kitada
Publikováno v:
Journal of lipid research. 63(6)
Diverse molecular species of sulfatide with differences in FA lengths, unsaturation degrees, and hydroxylation statuses are expressed in the kidneys. However, the physiological functions of specific sulfatide species in the kidneys are unclear. Here,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0127703bacd62e312d9618bbbf550887
https://pubmed.ncbi.nlm.nih.gov/35439525
https://pubmed.ncbi.nlm.nih.gov/35439525
Publikováno v:
Journal of Neuroscience Research. 98:325-337
Injured optic nerves induce death in almost all retinal ganglion cells (RGC) and cause a loss of axons. To date, we have studied injured RGC axon regeneration by using a traumatic optic nerve injury (TONI) rodent model, and we revealed that axonal re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd261368a7540c3c7575ddc9c41cd83b
https://doi.org/10.1002/jnr.24500
https://doi.org/10.1002/jnr.24500
Autor:
Nobuaki Shirai, Daijiro Yanagisawa, Koichi Hirao, Hiroyasu Taguchi, Keizo Gamo, Takami Tomiyama, Yukie Hirahara, Ikuo Tooyama, Masaaki Kitada, Takayuki Sogabe, Tomoko Kato
Publikováno v:
Biomaterials. 270
The accumulation of β-amyloid (Aβ) aggregates in the brain occurs early in the progression of Alzheimer's disease (AD), and non-fibrillar soluble Aβ oligomers are particularly neurotoxic. During binding to Aβ fibrils, curcumin, which can exist in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2091b98b39b8b6ff7b98cb94c18c372d
https://pubmed.ncbi.nlm.nih.gov/33540171
https://pubmed.ncbi.nlm.nih.gov/33540171