Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Keaton S. Karlinsey"'
Autor:
Antoine Ménoret, Federica Agliano, Timofey A. Karginov, Keaton S. Karlinsey, Beiyan Zhou, Anthony T. Vella
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
MicroRNA-150 (miR-150) has been shown to play a general role in the immune system, but very little is known about its role on CD4+ T cell responses. During T cell responses against superantigen Staphylococcal Enterotoxin A, miR-150 expression was dow
Externí odkaz:
https://doaj.org/article/7d33ae2362004c1e8e8efe8f3aca50db
Autor:
Ryan L Wood, Keaton S Karlinsey, Austin D Thompson, Mark N Rigby, Greggory D Boatright, William G Pitt, Beverly L Roeder, Scott C Steffensen, Alonzo D Cook
Publikováno v:
Neural Regeneration Research, Vol 13, Iss 5, Pp 846-853 (2018)
Schwann cells play a major role in helping heal injured nerves. They help clear debris, produce neurotrophins, upregulate neurotrophin receptors, and form bands of Büngner to guide the healing nerve. But nerves do not always produce enough neurotrop
Externí odkaz:
https://doaj.org/article/0a4e5e1995e9402484ff6c394745ed17
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-11 (2020)
Scientific Reports
Scientific Reports
Sterile stimuli can trigger inflammatory responses, and in some cases can lead to a variety of acute or chronic diseases. In this study, we hypothesize that a benzimidazole inhibitor may be used as a therapeutic in the treatment of sterile inflammati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f14a5c13aff7b3bc4a75e9c728a68c4c
http://link.springer.com/article/10.1038/s41598-020-68985-1
http://link.springer.com/article/10.1038/s41598-020-68985-1
Autor:
Federica Agliano, Antoine Ménoret, Keaton S. Karlinsey, Michael Ragazzi, Timofey Karginov, Anthony T. Vella
Publikováno v:
The Journal of Immunology. 204:144.21-144.21
Interleukin-1 receptor-associated kinase (IRAK) 4 is a serine/threonine kinase involved in TLR/IL-1R responses. Upon TLR/IL-1R activation, autophosphorylation of IRAK4 induces proinflammatory gene transcription. In this study we hypothesize that the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bfeb2bb24c6cc1878e27ca0d939d2dd9
https://doi.org/10.4049/jimmunol.204.supp.144.21
https://doi.org/10.4049/jimmunol.204.supp.144.21
Autor:
Goutham Pattabiraman, Keaton S. Karlinsey, Andrei E. Medvedev, Michael Murphy, Federica Agliano
IL-1 receptor-associated kinase (IRAK) 4 is a central enzyme of the TLR pathways. This study tested the hypothesis that IRAK4 kinase activity is prerequisite for regulating innate immunity during infections with intracellular bacteria. To this end, w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6dd167bfa132dc93250be8ab174818ac
https://europepmc.org/articles/PMC6162151/
https://europepmc.org/articles/PMC6162151/
