Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Kavi Mehta"'
Publikováno v:
Toxics, Vol 12, Iss 7, p 484 (2024)
Obesity and environmental toxins are risk factors for breast cancer; however, there is limited knowledge on how these risk factors interact to promote breast cancer. Acrylamide, a probable carcinogen and obesogen, is a by-product in foods prevalent i
Externí odkaz:
https://doaj.org/article/7e135dc7564049299010a88029687bf9
Autor:
Kavi Mehta, Laimonis Laimins
Publikováno v:
mBio, Vol 9, Iss 1 (2018)
ABSTRACT High-risk human papillomaviruses (HPVs) activate the ataxia telangiectasia mutated-dependent (ATM) DNA damage response as well as the ataxia telangiectasia mutated-dependent DNA-related (ATR) pathway in the absence of external DNA damaging a
Externí odkaz:
https://doaj.org/article/9502fc66e24f4061a74da84bcc4edd39
Publikováno v:
PLoS Pathogens, Vol 11, Iss 4, p e1004763 (2015)
Human papillomaviruses infect stratified epithelia and link their productive life cycle to the differentiation state of the host cell. Productive viral replication or amplification is restricted to highly differentiated suprabasal cells and is depend
Externí odkaz:
https://doaj.org/article/8dc8923d65884b18a3708fe4b03b3d1a
Autor:
Laimonis A. Laimins, Kavi Mehta
Publikováno v:
Viruses and Human Cancer ISBN: 9783030573614
Human papillomaviruses (HPVs) are small DNA viruses that infect basal epithelial cells and are the causative agents of cervical, anogenital, as well as oral cancers. High-risk HPVs are responsible for nearly half of all virally induced cancers. Viral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dc36d18977ca237502839fc4a24b2b8e
https://doi.org/10.1007/978-3-030-57362-1_7
https://doi.org/10.1007/978-3-030-57362-1_7
Autor:
Kavi, Mehta, Laimonis, Laimins
Publikováno v:
Recent results in cancer research. Fortschritte der Krebsforschung. Progres dans les recherches sur le cancer. 217
Human papillomaviruses (HPVs) are small DNA viruses that infect basal epithelial cells and are the causative agents of cervical, anogenital, as well as oral cancers. High-risk HPVs are responsible for nearly half of all virally induced cancers. Viral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::94837372e018110aa4199d5cba2e79a7
https://pubmed.ncbi.nlm.nih.gov/33200365
https://pubmed.ncbi.nlm.nih.gov/33200365
Publikováno v:
Journal of Virology. 85:8852-8862
Human papillomaviruses (HPVs) are the causative agents of several important genital and other mucosal cancers. The HPV16 E7 gene encodes a viral oncogene that is necessary for the continued growth of cancer cells, but its role in the normal, differen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4a064250be9d3cfbe8464433274adc9d
https://doi.org/10.1128/jvi.00664-11
https://doi.org/10.1128/jvi.00664-11
Publikováno v:
PLoS Pathogens
PLoS Pathogens, Vol 11, Iss 4, p e1004763 (2015)
PLoS Pathogens, Vol 11, Iss 4, p e1004763 (2015)
Human papillomaviruses infect stratified epithelia and link their productive life cycle to the differentiation state of the host cell. Productive viral replication or amplification is restricted to highly differentiated suprabasal cells and is depend
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6c07051257183972863069428b30abf2
http://europepmc.org/articles/PMC4395367
http://europepmc.org/articles/PMC4395367
Amplification of human papillomaviruses (HPV) is dependent on the ATM DNA damage pathway. In cells with impaired p53 activity, DNA damage repair requires the activation of p38MAPK along with MAPKAP kinase 2 (MK2). In HPV-positive cells, phosphorylati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ea39d38017a089efb5f17d78face1b3
https://europepmc.org/articles/PMC4300735/
https://europepmc.org/articles/PMC4300735/
Publikováno v:
Journal of virology. 86(17)
Human papillomaviruses (HPV) activate the ataxia telangiectasia mutated (ATM)-dependent DNA damage response to induce viral genome amplification upon epithelial differentiation. Our studies show that along with members of the ATM pathway, HPV protein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b97deec9b898f73277527be03f42defe
https://pubmed.ncbi.nlm.nih.gov/22740399
https://pubmed.ncbi.nlm.nih.gov/22740399