Zobrazeno 1 - 10
of 97
pro vyhledávání: '"Katsuya Kami"'
Autor:
Kohei Minami, Katsuya Kami, Yukihide Nishimura, Makoto Kawanishi, Kyosuke Imashiro, Takuma Kami, Shogo Habata, Emiko Senba, Yasunori Umemoto, Fumihiro Tajima
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-14 (2023)
Abstract The exact mechanism of exercise-induced hypoalgesia (EIH) in exercise therapy to improve chronic pain has not been fully clarified. Recent studies have suggested the importance of the ventral hippocampus (vHPC) in inducing chronic pain. We i
Externí odkaz:
https://doaj.org/article/f3b9ffdb525d4f498400362cd050ad7b
Autor:
Emiko Senba, Katsuya Kami
Publikováno v:
Neurobiology of Pain, Vol 14, Iss , Pp 100143- (2023)
We are exposed to various external and internal threats which might hurt us. The role of taking flexible and appropriate actions against threats is played by “the limbic system” and at the heart of it there is the ventral tegmental area and nucle
Externí odkaz:
https://doaj.org/article/069967b25f5646069b285612045e6ddd
Autor:
Makoto Kawanishi, Katsuya Kami, Yukihide Nishimura, Kohei Minami, Emiko Senba, Yasunori Umemoto, Tokio Kinoshita, Fumihiro Tajima
Publikováno v:
Physiological Reports, Vol 10, Iss 19, Pp n/a-n/a (2022)
Abstract Moderate‐intensity exercise performed during wound healing has been reported to decrease inflammatory cytokines and chemokines and accelerate wound healing. However, its effect on macrophage phenotype and the mechanism by which exercise ac
Externí odkaz:
https://doaj.org/article/75c9cf27966a4262a3c9f6b754bde4d8
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 5, p 2886 (2022)
It is well known that exercise produces analgesic effects (exercise-induced hypoalgesia (EIH)) in animal models and chronic pain patients, but the brain mechanisms underlying these EIH effects, especially concerning the emotional aspects of pain, are
Externí odkaz:
https://doaj.org/article/3d05e7df24844509afcd58ab553e2ff2
Autor:
Emiko Senba, Katsuya Kami
Publikováno v:
Neurobiology of Pain, Vol 1, Iss , Pp 6-15 (2017)
Physical exercise has been established as a low-cost, safe, and effective way to manage chronic intractable pain. We investigated the underlying mechanisms of exercise-induced hypoalgesia (EIH) using a mouse model of neuropathic pain (NPP). Epigeneti
Externí odkaz:
https://doaj.org/article/7503b8651ca34ff6a7d3d73a9bfafb08
Autor:
Kohei Minami, Katsuya Kami, Yukihide Nishimura, Makoto Kawanishi, Kyosuke Imashiro, Takuma Kami, Shogo Habata, Emiko Senba, Yasunori Umemoto, Fumihiro Tajima
The exact mechanism of exercise-induced hypoalgesia (EIH) in exercise therapy to improve chronic pain has not been fully clarified. Recent studies have suggested the importance of the ventral hippocampus (vHPC) in inducing chronic pain. We investigat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c1b24a1921d6ceef4e87cb9f0e1a1fd9
https://doi.org/10.21203/rs.3.rs-2173132/v1
https://doi.org/10.21203/rs.3.rs-2173132/v1
Publikováno v:
PAIN RESEARCH. 35:80-91
Publikováno v:
Molecular Pain. 2016, Vol. 12, p1-14. 14p. 5 Graphs.
Publikováno v:
Scientific Reports, Vol 8, Iss 1, Pp 1-14 (2018)
Scientific Reports
Scientific Reports
Ventral tegmental area (VTA) dopamine (DA) neurons are the primary source of dopamine in target structures that constitute the mesolimbic reward system. Previous studies demonstrated that voluntary wheel running (VWR) by neuropathic pain (NPP) model
Autor:
Katsuya Kami, Emiko Senba
Publikováno v:
Neurobiology of Pain
Neurobiology of Pain, Vol 1, Iss, Pp 6-15 (2017)
Neurobiology of Pain, Vol 1, Iss, Pp 6-15 (2017)
Graphical abstract
Highlights • Activation of mesolimbic dopamine system underlies exercise-induced hypoalgesia. • Interaction between mesolimbic system and hypothalamus determines physical activity. • Changing the lifestyle inactive to ac
Highlights • Activation of mesolimbic dopamine system underlies exercise-induced hypoalgesia. • Interaction between mesolimbic system and hypothalamus determines physical activity. • Changing the lifestyle inactive to ac