Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Katsuki Kobayashi"'
Publikováno v:
Nihon Toseki Igakkai Zasshi. 56:117-120
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a7744f0c8102aa8478fe56e5f68e0604
https://doi.org/10.4009/jsdt.56.117
https://doi.org/10.4009/jsdt.56.117
Autor:
Katsuki Kobayashi, Satoshi Tsujimoto
The discrete Toda lattice preserves the eigenvalues of tridiagonal matrices, and convergence of dependent variables to the eigenvalues can be proved under appropriate conditions. We show that the ultradiscrete Toda lattice preserves invariant factors
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad59823a83a55ea5a31d51a5e630b91a
http://arxiv.org/abs/2012.13068
http://arxiv.org/abs/2012.13068
Publikováno v:
Communications in Computer and Information Science ISBN: 9783030507282
HCI (39)
HCI (39)
In recent years, VR devices such as PlayStation VR, HTC Vive, and Oculus Rift have become widespread, making it possible to easily experience Virtual Reality. There are contents that can get a high immersive feeling by synchronizing the motion of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::952b76af4f33fcbe477b5ad657772665
https://doi.org/10.1007/978-3-030-50729-9_29
https://doi.org/10.1007/978-3-030-50729-9_29
Autor:
Katsuki Kobayashi
Publikováno v:
Journal of Physics A: Mathematical and Theoretical. 54:455203
We derive a nonautonomous discrete analogue of the elementary Toda orbits via a spectral transformation of certain biorthogonal Laurent polynomials. We obtain particular solutions of the system by using the theory of biorthogonal polynomials. The obt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::166c7f0626b645e3650fa024ef6b421a
https://doi.org/10.1088/1751-8121/ac2cff
https://doi.org/10.1088/1751-8121/ac2cff
Autor:
Jing Zhou, Shigeo Horie, Motoko Chiga, Sei Sasaki, Yuichi Inoue, Shinichi Uchida, Katsuki Kobayashi, Xuefeng Su, Kenichi Ishibashi, Eisei Sohara, Tatemitsu Rai
Publikováno v:
Journal of the American Society of Nephrology. 25:2789-2799
We previously reported that disruption of the aquaporin-11 (AQP11) gene in mice resulted in cystogenesis in the kidney. In this study, we aimed to clarify the mechanism of cystogenesis in AQP11(-/-) mice. To enable the analyses of AQP11 at the protei
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f873a665cb8ce6205169aa0df2015a88
https://doi.org/10.1681/asn.2013060614
https://doi.org/10.1681/asn.2013060614
Publikováno v:
ITE Transactions on Media Technology and Applications. 1:333-342
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::da3d17b750a5e47e7557fbda38820253
https://doi.org/10.3169/mta.1.333
https://doi.org/10.3169/mta.1.333
Autor:
Ayaka Andoo, Kuniaki Takata, Sei Sasaki, Shigeo Ookawara, Mariko Shimono, Eiji Kusano, Asako Matsuki, Toshiyuki Matsuzaki, Katsuki Kobayashi, Mariko Hara-Chikuma, Alan S. Verkman, Tadashi Yamamoto, Yoshiyuki Morishita, Masahiro Ikeda, Kenichi Ishibashi
Publikováno v:
Molecular and Cellular Biology. 25:7770-7779
Aquaporin-11 (AQP11) has been identified with unusual pore-forming NPA (asparagine-proline-alanine) boxes, but its function is unknown. We investigated its potential contribution to the kidney. Immunohistochemistry revealed that AQP11 was localized i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1552042c45df932d1b0297e5a463860a
https://doi.org/10.1128/mcb.25.17.7770-7779.2005
https://doi.org/10.1128/mcb.25.17.7770-7779.2005
Disease-causing mutant WNK4 increases paracellular chloride permeability and phosphorylates claudins
Autor:
Tatemitsu Rai, Kozue Yamauchi, Eisei Sohara, Tomohiro Itoh, Tatsunori Suzuki, Atsushi Hayama, Shinichi Uchida, Katsuki Kobayashi, Shin Suda, Sei Sasaki
Publikováno v:
Proceedings of the National Academy of Sciences. 101:4690-4694
Mutations in the WNK4 gene cause pseudohypoaldosteronism type II (PHAII), an autosomal-dominant disorder of hyperkalemia and hypertension. The target molecules of this putative kinase and the molecular mechanisms by which the mutations cause the phen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7921d0b3dcac84a25978c071dc044f5
https://doi.org/10.1073/pnas.0306924101
https://doi.org/10.1073/pnas.0306924101
Autor:
Yasuo Uchiyama, Shinichi Uchida, Yujiro Kida, Atsushi Hayama, Sei Sasaki, Junji Ezaki, Masato Koike, Fumiaki Marumo, Masaki Noda, Tatemitsu Rai, Eiki Kominami, Momono Yoshikawa, Katsuki Kobayashi
Publikováno v:
Genes to Cells. 7:597-605
Background: CLC-3 is a member of the CLC chloride channel family and is widely expressed in mammalian tissues. To determine the physiological role of CLC-3, we generated CLC-3-deficient mice (Clcn3–/–) by targeted gene disruption. Results: Togeth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::609a51cff4f899c0f4309af25f28f4da
https://doi.org/10.1046/j.1365-2443.2002.00539.x
https://doi.org/10.1046/j.1365-2443.2002.00539.x
Publikováno v:
Histochemistry and Cell Biology. 116:49-56
CLC-K1, a kidney-specific chloride channel, has been demonstrated to be involved in the urine concentration mechanism. Here, we investigated the developmental expression of CLC-K1 in the rat kidney. Using immunohistochemistry, we showed that CLC-K1 w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a9cf66b9b4f6c5849299e1c79c6947c
https://doi.org/10.1007/s004180100294
https://doi.org/10.1007/s004180100294