Zobrazeno 1 - 10
of 117
pro vyhledávání: '"Katsuhisa, Masaki"'
Autor:
Asuka Tani, Shinsuke Mizutani, Mitsuru Watanabe, Takashi Irie, Katsuhisa Masaki, Noriko Isobe, Haruhiko Kashiwazaki
Publikováno v:
BMC Oral Health, Vol 24, Iss 1, Pp 1-5 (2024)
Abstract Background Isaacs’ syndrome, also known as neuromyotonia or peripheral nerve hyperexcitability, is a rare disorder that affects the peripheral nervous system. Clinical findings include cramps, fasciculations, and myokymia; however, there a
Externí odkaz:
https://doaj.org/article/3568dda25999446ab3ca2038d7f25780
Autor:
Ezgi Ozdemir Takase, Ryo Yamasaki, Satoshi Nagata, Mitsuru Watanabe, Katsuhisa Masaki, Hiroo Yamaguchi, Jun-ichi Kira, Hideyuki Takeuchi, Noriko Isobe
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-23 (2024)
Abstract In chronic stages of multiple sclerosis (MS) and its animal model, experimental autoimmune encephalitis (EAE), connexin (Cx)43 gap junction channel proteins are overexpressed because of astrogliosis. To elucidate the role of increased Cx43,
Externí odkaz:
https://doaj.org/article/677e0f459eba49d6aa9029b65a57d82b
Autor:
Satoshi Nagata, Ryo Yamasaki, Ezgi Ozdemir Takase, Kotaro Iida, Mitsuru Watanabe, Katsuhisa Masaki, Marion Heleen Cathérine Wijering, Hiroo Yamaguchi, Jun-ichi Kira, Noriko Isobe
Publikováno v:
Biology, Vol 12, Iss 9, p 1217 (2023)
We previously reported a novel secondary progressive multiple sclerosis (SPMS) model, progressive experimental autoimmune encephalomyelitis (pEAE), in oligodendroglia-specific Cx47-inducible conditional knockout (Cx47 icKO) mice. Based on our prior s
Externí odkaz:
https://doaj.org/article/90bddc8b7a7146269bca5792f1f689bb
Autor:
Hayato Une, Ryo Yamasaki, Satoshi Nagata, Hiroo Yamaguchi, Yuko Nakamuta, Ulfa Camelia Indiasari, Yiwen Cui, Koji Shinoda, Katsuhisa Masaki, Magdalena Götz, Jun-ichi Kira
Publikováno v:
Journal of Neuroinflammation, Vol 18, Iss 1, Pp 1-19 (2021)
Abstract Background Brain astroglia are activated preceding the onset of experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). We characterized the effects of brain astroglia on spinal cord inflammation, focusin
Externí odkaz:
https://doaj.org/article/37938b5b62a04f64beb4ea1d43370c05
Autor:
Mitsuru Watanabe, Yuri Nakamura, Shinya Sato, Masaaki Niino, Hikoaki Fukaura, Masami Tanaka, Hirofumi Ochi, Takashi Kanda, Yukio Takeshita, Takanori Yokota, Yoichiro Nishida, Makoto Matsui, Shigemi Nagayama, Susumu Kusunoki, Katsuichi Miyamoto, Masanori Mizuno, Izumi Kawachi, Etsuji Saji, Takashi Ohashi, Shun Shimohama, Shin Hisahara, Kazutoshi Nishiyama, Takahiro Iizuka, Yuji Nakatsuji, Tatsusada Okuno, Kazuhide Ochi, Akio Suzumura, Ken Yamamoto, Yuji Kawano, Shoji Tsuji, Makoto Hirata, Ryuichi Sakate, Tomonori Kimura, Yuko Shimizu, Akiko Nagaishi, Kazumasa Okada, Fumie Hayashi, Ayako Sakoda, Katsuhisa Masaki, Koji Shinoda, Noriko Isobe, Takuya Matsushita, Jun-ichi Kira
Publikováno v:
Scientific Reports, Vol 11, Iss 1, Pp 1-16 (2021)
Abstract HLA genotype-clinical phenotype correlations are not established for multiple sclerosis (MS) and neuromyelitis optica spectrum disorders (NMOSD). We studied HLA-DRB1/DPB1 genotype–phenotype correlations in 528 MS and 165 NMOSD cases using
Externí odkaz:
https://doaj.org/article/67db0d0897834f4e92a7985435c2ebfc
Autor:
Yu Hashimoto, Ryo Yamasaki, Senri Ko, Eriko Matsuo, Yuko Kobayakawa, Katsuhisa Masaki, Dai Matsuse, Noriko Isobe
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 24, p 16046 (2022)
Connexin 30 (Cx30), which forms gap junctions between astrocytes, regulates cell adhesion and migration, and modulates glutamate transport. Cx30 is upregulated on activated astroglia in central nervous system inflammatory lesions, including spinal co
Externí odkaz:
https://doaj.org/article/9c46d7f79a01481d9c283de359581a39
Autor:
Takuya Matsushita, Katsuhisa Masaki, Noriko Isobe, Shinya Sato, Ken Yamamoto, Yuri Nakamura, Mitsuru Watanabe, Toshihiko Suenaga, Jun‐ichi Kira, the Japan Multiple Sclerosis Genetic Consortium
Publikováno v:
Annals of Clinical and Translational Neurology, Vol 7, Iss 11, Pp 2082-2093 (2020)
Abstract Objective To identify genetic factors associated with susceptibility to and clinical features of neuromyelitis optica spectrum disorders (NMOSD). Methods Genome‐wide single nucleotide polymorphism (SNP) genotyping was conducted in 211 Japa
Externí odkaz:
https://doaj.org/article/e3a40113f55b4a8e8e11e45e8dd028f0
Autor:
Toshiki Okadome, Hajime Takeuchi, Takahiro Yamaguchi, Takahiko Mukaino, Hidenori Ogata, Katsuhisa Masaki, Hiroshi Shigeto, Noriko Isobe
Publikováno v:
Epilepsy & Behavior Reports, Vol 19, Iss , Pp 100543- (2022)
Exercise-induced reflex seizures are a rare form of reflex seizures that are exclusively induced by a specific type of exercise. Many patients with exercise-induced reflex seizures exhibit drug-resistance, and are therefore advised to avoid the trigg
Externí odkaz:
https://doaj.org/article/4ab5a24fe26d41fea9a759b5d8334bb6
Autor:
Guzailiayi Maimaitijiang, Mitsuru Watanabe, Koji Shinoda, Noriko Isobe, Yuri Nakamura, Katsuhisa Masaki, Takuya Matsushita, Yasunobu Yoshikai, Jun-ichi Kira
Publikováno v:
Journal of Neuroinflammation, Vol 16, Iss 1, Pp 1-15 (2019)
Abstract Background We previously reported that Vδ2+Vγ9+ γδ T cells were significantly decreased in multiple sclerosis (MS) patients without disease-modifying therapies (untreated MS) and were negatively correlated with Expanded Disability Status
Externí odkaz:
https://doaj.org/article/ce6e9f698717401ab1d341926977e6a9
Autor:
Yukino Miyachi, Takayuki Fujii, Ryo Yamasaki, Daisuke Tsuchimoto, Kyoko Iinuma, Ayako Sakoda, Shoko Fukumoto, Takuya Matsushita, Katsuhisa Masaki, Noriko Isobe, Yusaku Nakabeppu, Jun-ichi Kira
Publikováno v:
Frontiers in Neurology, Vol 12 (2021)
Multiple sclerosis (MS), the most prevalent inflammatory disease of the central nervous system (CNS), is characterized by damaged to myelin sheaths and oligodendrocytes. Because MS patients have variable clinical courses and disease severities, it is
Externí odkaz:
https://doaj.org/article/543075fd8a9a4863a32b53b50542e7de