Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Katja Metzger"'
Autor:
Michael Gramlich, Luna Simona Pane, Qifeng Zhou, Zhifen Chen, Marta Murgia, Sonja Schötterl, Alexander Goedel, Katja Metzger, Thomas Brade, Elvira Parrotta, Martin Schaller, Brenda Gerull, Ludwig Thierfelder, Annemieke Aartsma‐Rus, Siegfried Labeit, John J Atherton, Julie McGaughran, Richard P Harvey, Daniel Sinnecker, Matthias Mann, Karl‐Ludwig Laugwitz, Meinrad Paul Gawaz, Alessandra Moretti
Publikováno v:
EMBO Molecular Medicine, Vol 7, Iss 5, Pp 562-576 (2015)
Abstract Frameshift mutations in the TTN gene encoding titin are a major cause for inherited forms of dilated cardiomyopathy (DCM), a heart disease characterized by ventricular dilatation, systolic dysfunction, and progressive heart failure. To date,
Externí odkaz:
https://doaj.org/article/7c654a80f21048ac9a078d405a3d0a79
Autor:
Madhumita Chatterjee, Peter Seizer, Dominik Rath, Katja Metzger, Anna Fotinos, Oliver Borst, Tobias Geisler, Evi Schmid, Meinrad Gawaz, Florian Lang, S Hoffmann, Sebastian Vogel, Britta Walker, Patrick Münzer
Publikováno v:
American Journal of Physiology-Cell Physiology. 307:C920-C927
Sphingosine 1-phosphate (S1P) is a powerful regulator of platelet formation. Enzymes generating S1P include sphingosine kinase 1. The present study thus explored the role of sphingosine kinase 1 in platelet formation and function. Activation-dependen
Autor:
Daniel Sinnecker, Richard P. Harvey, Elvira Immacolata Parrotta, Martin Schaller, Brenda Gerull, Zhifen Chen, Marta Murgia, Sonja Schötterl, Katja Metzger, Annemieke Aartsma-Rus, Karl-Ludwig Laugwitz, Michael Gramlich, Siegfried Labeit, Meinrad Gawaz, Julie McGaughran, Thomas Brade, John Atherton, Qifeng Zhou, Alessandra Moretti, Luna Simona Pane, Matthias Mann, Ludwig Thierfelder, Alexander Goedel
Publikováno v:
EMBO Molecular Medicine
EMBO Molecular Medicine, 7(5), 562-576
ResearcherID
EMBO MOLECULAR MEDICINE
EMBO Molecular Medicine, 7(5), 562-576
ResearcherID
EMBO MOLECULAR MEDICINE
Frameshift mutations in the TTN gene encoding titin are a major cause for inherited forms of dilated cardiomyopathy (DCM), a heart disease characterized by ventricular dilatation, systolic dysfunction, and progressive heart failure. To date, there ar
Autor:
Dietmar Kuhl, Ioana Alesutan, Sebastian Vogel, Florian Lang, Malte Schaub, Britta Walker, Oliver Borst, Tanja Schoenberger, Meinrad Gawaz, Anja T. Umbach, Peter Seizer, Dominik Rath, Evi Schmid, Iris Müller, Katja Metzger, Tobias Geisler, Jose Rodriguez, Jakob Voelkl, Patrick Münzer
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 35(3)
Objective— Atherosclerosis, an inflammatory disease of arterial vessel walls, requires migration and matrix metalloproteinase (MMP)-9–dependent invasion of monocytes/macrophages into the vascular wall. MMP-9 expression is stimulated by transcript
Autor:
Madhumita Chatterjee, Katja Metzger, Oliver Borst, Tobias Geisler, Meinrad Gawaz, Tanja Schönberger, Peter Seizer, Harald F. Langer, Sebastian Vogel, Tilman E. Schäffer, Florian Lang, Johannes Rheinlaender
Publikováno v:
Biomaterials. 36
Targeted contrast-enhanced ultrasound (CEU) using microbubble agents is a promising non-invasive imaging technique to evaluate atherosclerotic lesions. In this study, we decipher the diagnostic and therapeutic potential of targeted-CEU with soluble g
Autor:
Katja Metzger, Meinrad Gawaz, Thorsten Trapp, Rüdiger V. Sorg, Sebastian Vogel, Corinna Peters, Roland Meisel, Walter Däubener, Verena Börger, M Förster, P Liebfried, Johannes C. Fischer
Publikováno v:
Cell death and differentiation. 22(7)
Tissue damage due to apoptotic or necrotic cell death typically initiates distinct cellular responses, leading either directly to tissue repair and regeneration or to immunological processes first, to clear the site, for example, of potentially damag
Autor:
Peter Seizer, Harald F. Langer, Madhumita Chatterjee, Oliver Borst, Susanne Schumann, Sebastian Vogel, Florian Lang, Katja Metzger, Andreas F. Mack, Tobias Geisler, Iris Müller, Rüdiger V. Sorg, Hans-Jörg Bühring, Meinrad Gawaz
Recruitment of mesenchymal stem cells (MSC) following cardiac injury, such as myocardial infarction, plays a critical role in tissue repair and may contribute to myocardial recovery. However, the mechanisms that regulate migration of MSC to the site
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6221a5ba9af2d71ea34a16fc39b11c7a
https://europepmc.org/articles/PMC4036247/
https://europepmc.org/articles/PMC4036247/