Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Katja Balazs"'
Autor:
Stefan Bartl, Yuanyun Xie, Nalini Potluri, Ratnesh Kesineni, Katlin Hencak, Louisa Dal Cengio, Katja Balazs, Abid Oueslati, Michela Parth, Nina Salhat, Alberto Siddu, Oskar Smrzka, Francesca Cicchetti, Günther Staffler, Michael R. Hayden, Amber L. Southwell
Publikováno v:
Neurobiology of Disease, Vol 193, Iss , Pp 106444- (2024)
Externí odkaz:
https://doaj.org/article/9f127a6b6c0642d8b90cf4f316b4916a
Autor:
Stefan Bartl, Yuanyun Xie, Nalini Potluri, Ratnesh Kesineni, Katlin Hencak, Louisa Dal Cengio, Katja Balazs, Abid Oueslati, Michela Parth, Nina Salhat, Alberto Siddu, Oskar Smrzka, Francesca Cicchetti, Günther Straffler, Michael R. Hayden, Amber L. Southwell
Publikováno v:
Neurobiology of Disease, Vol 190, Iss , Pp 106376- (2024)
In Huntington disease (HD), the mutant huntingtin (mtHTT) protein is the principal cause of pathological changes that initiate primarily along the cortico-striatal axis. mtHTT is ubiquitously expressed and there is, accordingly, growing recognition t
Externí odkaz:
https://doaj.org/article/4d07a71b09d34d4a99a06f94d8778757
Autor:
Stefan Bartl, Abid Oueslati, Amber L. Southwell, Alberto Siddu, Michela Parth, Linda Suzanne David, Alexander Maxan, Nina Salhat, Markus Burkert, Andreas Mairhofer, Theresa Friedrich, Halyna Pankevych, Katja Balazs, Guenther Staffler, Michael R. Hayden, Francesca Cicchetti, Oskar W. Smrzka
Publikováno v:
Neurobiology of Disease, Vol 141, Iss , Pp 104943- (2020)
Huntington's disease (HD) is caused by a highly polymorphic CAG trinucleotide expansion in the gene encoding for the huntingtin protein (HTT). The resulting mutant huntingtin protein (mutHTT) is ubiquitously expressed but also exhibits the ability to
Externí odkaz:
https://doaj.org/article/7a2a9cc730a442b79298549f98f442c9
Autor:
Borja Mateos, Judith Konrat, Katja Balazs, Robert Konrat, Guenther Staffler, Marco Sealey-Cardona
Publikováno v:
Angewandte Chemie (International Ed. in English)
Interactions of transmembrane receptors with their extracellular ligands are essential for cellular communication and signaling and are therefore a major focus in drug discovery programs. The transition from in vitro to live cell interaction studies,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::20cf0105fc87701a43d536b33e27744d
https://doi.org/10.1002/anie.201913585
https://doi.org/10.1002/anie.201913585
Autor:
Halyna Pankevych, Stefan Bartl, Amber L. Southwell, Michela Parth, Theresa Friedrich, Alexander Maxan, Michael R. Hayden, Francesca Cicchetti, Guenther Staffler, Andreas Mairhofer, Alberto Siddu, Nina Salhat, Abid Oueslati, Katja Balazs, Markus Burkert, Oskar W. Smrzka, Linda Suzanne David
Publikováno v:
Neurobiology of Disease, Vol 141, Iss, Pp 104943-(2020)
Huntington's disease (HD) is caused by a highly polymorphic CAG trinucleotide expansion in the gene encoding for the huntingtin protein (HTT). The resulting mutant huntingtin protein (mutHTT) is ubiquitously expressed but also exhibits the ability to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::960fa749199bcd14ff701a186072beed
https://pubmed.ncbi.nlm.nih.gov/32407769
https://pubmed.ncbi.nlm.nih.gov/32407769