Výsledky vyhledávání - "Kathryn Repp"

Akademický článek

Circulating levels of mitochondrial uncoupling protein 2, but not prohibitin, are lower in humans with type 2 diabetes and correlate with brachial artery flow-mediated dilation

Autor: Mamatha Kakarla, Venkata K. Puppala, Sudhi Tyagi, Amberly Anger, Kathryn Repp, Jingli Wang, Rong Ying, Michael E. Widlansky

Publikováno v: Cardiovascular Diabetology, Vol 18, Iss 1, Pp 1-11 (2019)

Abstract Background Excessive reactive oxygen species from endothelial mitochondria in type 2 diabetes individuals (T2DM) may occur through multiple related mechanisms, including production of mitochondrial reactive oxygen species (mtROS), inner mito

Externí odkaz: https://doaj.org/article/dfd4ba76cce04178a547e91a404aeabe

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Circulating levels of mitochondrial uncoupling protein 2, but not prohibitin, are lower in humans with type 2 diabetes and correlate with brachial artery flow-mediated dilation

Autor: Sudhi Tyagi, Amberly Anger, Jingli Wang, Kathryn Repp, Michael E. Widlansky, Venkata K. Puppala, Mamatha Kakarla, Rong Ying

Publikováno v: Cardiovascular Diabetology
Cardiovascular Diabetology, Vol 18, Iss 1, Pp 1-11 (2019)

Background Excessive reactive oxygen species from endothelial mitochondria in type 2 diabetes individuals (T2DM) may occur through multiple related mechanisms, including production of mitochondrial reactive oxygen species (mtROS), inner mitochondrial

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::200342db22209f31d847a4424246e50f
https://pubmed.ncbi.nlm.nih.gov/31706320

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Human genotyping and an experimental model reveal NPR-C as a possible contributor to morbidity in coarctation of the aorta

Autor: Michael E. Mitchell, Kathryn Repp, Mary Krolikowski, Donna K. Mahnke, Kathleen Bazan, Jesse W. Gerringer, Brandon J. Wegter, Adrian Zietara, Mary Goetsch, Karl Stamm, Thomas J. Eddinger, Oleg Palygin, John F. LaDisa, Abdel A. Alli, Aoy Tomita-Mitchell

Publikováno v: Physiol Genomics

Coarctation of the aorta (CoA) is a common congenital cardiovascular (CV) defect characterized by a stenosis of the descending thoracic aorta. Treatment exists, but many patients develop hypertension (HTN). Identifying the cause of HTN is challenging

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cee1e34954427f7f8c8371d37c655128
https://pubmed.ncbi.nlm.nih.gov/31002586

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