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pro vyhledávání: '"Kathleen E McDougall"'
Autor:
C A Downs, Kathleen E McDougall, Cheryl M Woodley, John E Fauth, Robert H Richmond, Ariel Kushmaro, Stuart W Gibb, Yossi Loya, Gary K Ostrander, Esti Kramarsky-Winter
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e77173 (2013)
Coral bleaching is a significant contributor to the worldwide degradation of coral reefs and is indicative of the termination of symbiosis between the coral host and its symbiotic algae (dinoflagellate; Symbiodinium sp. complex), usually by expulsion
Externí odkaz:
https://doaj.org/article/3c6607e92ea2445f9b1d4842dc1884b9
Publikováno v:
Bone. 42:216-225
Bone morphogenetic protein 6 (BMP-6) is expressed by different skeletal cells including osteoblasts and growth plate chondrocytes, suggesting roles in bone formation and growth regulation. To address these possibilities, we examined whether cancellou
Autor:
Stuart W. Gibb, Craig A. Downs, Esti Kramarsky-Winter, Robert H. Richmond, Cheryl M. Woodley, Ariel Kushmaro, Yossi Loya, Kathleen E. McDougall, John E. Fauth, Gary K. Ostrander
Publikováno v:
PLoS ONE, Vol 8, Iss 12, p e77173 (2013)
United States National Ocean Service
PLoS ONE
United States National Ocean Service
PLoS ONE
Coral bleaching is a significant contributor to the worldwide degradation of coral reefs and is indicative of the termination of symbiosis between the coral host and its symbiotic algae (dinoflagellate; Symbiodinium sp. complex), usually by expulsion
Autor:
Mark Perry, Jonathan H Tobias, Shane M. Colley, Rachel L. Gibson, Kenneth S. Korach, Kathleen E. McDougall
Publikováno v:
Endocrinology. 144(5)
We examined whether estrogen receptor (ER)alpha is required for estrogen to stimulate cancellous bone formation in long bones of male mice. 17 beta-Estradiol (E(2)) was administered to ER alpha(-/-) male mice or wild-type (WT) littermate controls at
Autor:
J B Hodgin, O Smithies, J M Bright, Jonathan H Tobias, R L Gibson, Mark Perry, Shane M. Colley, Kathleen E. McDougall
Publikováno v:
Europe PubMed Central
We recently found that estrogen receptor (ER) antagonists prevent high-dose estrogen from inducing the formation of new cancellous bone within the medullary cavity of mouse long bones. In the present investigation, we studied the role of specific ER