Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Katharina Slavnitsch"'
Autor:
Michael Willmann, Barbara Peter, Katharina Slavnitsch, Daniela Berger, Nadine Witzeneder, Gabriele Stefanzl, Gregor Eisenwort, Daniel Ivanov, Irina Sadovnik, Emir Hadzijusufovic, Georg Greiner, Tina Bernthaler, Gregor Hoermann, Maik Dahlhoff, Thomas Rülicke, Peter Valent
Publikováno v:
Leukemia. 37:1162-1165
Autor:
Daniel Ivanov, Jelena D. Milosevic Feenstra, Irina Sadovnik, Harald Herrmann, Barbara Peter, Michael Willmann, Georg Greiner, Katharina Slavnitsch, Emir Hadzijusufovic, Thomas Rülicke, Maik Dahlhoff, Gregor Hoermann, Sigrid Machherndl‐Spandl, Gregor Eisenwort, Michael Fillitz, Thamer Sliwa, Maria‐Theresa Krauth, Peter Bettelheim, Wolfgang R. Sperr, Elisabeth Koller, Michael Pfeilstöcker, Heinz Gisslinger, Felix Keil, Robert Kralovics, Peter Valent
Publikováno v:
American Journal of Hematology. 98:770-783
Autor:
Daniela Berger, Alexandra Keller, Georg Greiner, Daniel Ivanov, Thomas Rülicke, Peter Bettelheim, Michael W. Deininger, Irina Sadovnik, Heinz Sill, Karin Bauer, Michael Willmann, Karoline V. Gleixner, Wolfgang R. Sperr, Gregor Eisenwort, Barbara Peter, Peter Valent, Gabriele Stefanzl, Katharina Slavnitsch, Klaus Geissler
Publikováno v:
Leukemia
Chronic myelomonocytic leukemia (CMML) is a stem cell-derived neoplasm characterized by dysplasia, uncontrolled expansion of monocytes and a substantial risk to transform to secondary acute myeloid leukemia (sAML). So far, little is known about CMML-
Autor:
Georg Greiner, Daniel Ivanov, Katharina Slavnitsch, Robert Spörk, Alexandra Keller, Michael Fillitz, Gregor Eisenwort, Klaus Geissler, Robert Kralovics, Michael Willmann, Jelena D. Milosevic Feenstra, Peter Valent, Michael Pfeilstöcker, Thamer Sliwa, Elisabeth Koller, Gabriele Stefanzl, Heinz Sill, Peter Bettelheim, Felix Keil, Sybille Hofer, Thomas Rülicke, Gregor Hoermann, Wolfgang R. Sperr, Barbara Peter
Publikováno v:
Blood. 136:53-53
The classical BCR-ABL1-negative myeloproliferative neoplasms (MPN) are characterized by over-production of myeloid cells, disease-related mutations in certain driver-genes (JAK2, CALR, MPL) and an increased risk to transform to secondary acute myeloi
Publikováno v:
Alzheimer's & Dementia. 6