Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Katharina Schulenburg"'
Publikováno v:
European heart journal. 41(16)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c50524558f2eb5d8ed248df3656a01f3
https://pubmed.ncbi.nlm.nih.gov/32318738
https://pubmed.ncbi.nlm.nih.gov/32318738
Autor:
Bernd Thiede, Frauke Zipp, Daniel Abankwa, Krishnaraj Rajalingam, Stefan Tenzer, Hajime Yurugi, Katharina Schulenburg, Tobias Bopp, Alexander Ulges, Maja Šolman, Ulrike Buehler
Publikováno v:
The EMBO journal. 37(16)
T helper (Th)17 cells represent a unique subset of CD4(+) T cells and are vital for clearance of extracellular pathogens including bacteria and fungi. However, Th17 cells are also involved in orchestrating autoimmunity. By employing quantitative surf
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1f3e0a3bdc56a0e2ecacc2c35c88638b
https://pubmed.ncbi.nlm.nih.gov/30049713
https://pubmed.ncbi.nlm.nih.gov/30049713
Autor:
Daniel Anthes, Katharina Schulenburg
Teller statt Tonne! Unglaublich, aber wahr: 313 Kilogramm genießbare Lebensmittel werden pro Sekunde in Deutschland weggeworfen. Kaum ist das Mindesthaltbarkeitsdatum des Joghurts überschritten oder der Apfel nicht mehr ganz so frisch, landen sie i
Autor:
Boris Macek, Katharina Schulenburg, Armelle Natsuo Takeda, Attila Reményi, Tripat Kaur Oberoi-Khanuja, Krishnaraj Rajalingam, Alejandro Carpy, Gábor Glatz, Rolf Peter Scholz
Publikováno v:
The EMBO Journal. 33:1784-1801
Mitogen-activated protein kinases (MAPKs) are highly conserved protein kinase modules, and they control fundamental cellular processes. While the activation of MAPKs has been well studied, little is known on the mechanisms driving their inactivation.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::26fa17105ec9acd0f57b276b5ff3d09b
https://doi.org/10.15252/embj.201487808
https://doi.org/10.15252/embj.201487808
Publikováno v:
Molecular & Cellular Oncology. 1:e969170
Recently, we revealed that ubiquitination of MEKK2 and MEKK3 by inhibitor of apoptosis proteins (IAPs) directly disrupts MEK5/ERK5 interaction and subsequently attenuates ERK5 activation. In addition, loss of XIAP promotes human myogenic differentiat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5015944b1bd8d408c4258210c86ae371
https://doi.org/10.4161/23723548.2014.969170
https://doi.org/10.4161/23723548.2014.969170