Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Katharina S. Rauch"'
Autor:
Sebastian J. Arnold, Laure Garnier, Katharina S. Rauch, Kristina Schachtrup, Sophie Laffont, Maria Brack, Ana Izcue, Jean-Charles Guéry, Ekaterina Lupar
Publikováno v:
The Journal of Immunology. 195:4742-4752
CD4+ T cells polarize into effector Th subsets characterized by signature transcription factors and cytokines. Although T-bet drives Th1 responses and represses the alternative Th2, Th17, and Foxp3+ regulatory T cell fates, the role of the T-bet–re
Autor:
Dietmar Pfeifer, Leandra Plappert, Miriam Hils, Könül Mammadzada, Christian Schachtrup, Kristina Schachtrup, Katharina S. Rauch, Christian Bohrer, Matthias Kirsch, Verónica I. Dumit, Jörn Dengjel, Sebastian Schildge, Lauriane Pous, Sabrina Pfurr
Publikováno v:
The EMBO Journal. 34:2804-2819
Adult neural stem/precursor cells (NSPCs) of the subventricular zone (SVZ) are an endogenous source for neuronal replacement in CNS disease. However, adult neurogenesis is compromised after brain injury in favor of a glial cell fate, which is mainly
Autor:
Peter Aichele, Christian Schachtrup, Alexandra J. Menner, Katharina S. Rauch, Kristina Schachtrup, Hanspeter Pircher
Publikováno v:
The Journal of Immunology. 195:2103-2114
Sustained Ag persistence in chronic infection results in a deregulated CD8+ T cell response that is characterized by T cell exhaustion and cell death of Ag-specific CD8+ T cells. Yet, the underlying transcriptional mechanisms regulating CD8+ T cell e
Autor:
Susana Minguet, Christian Schachtrup, Miriam Hils, Mikael Sigvardsson, Katharina S. Rauch, Peter Aichele, Kristina Schachtrup, Alexandra J. Menner
Publikováno v:
Oncotarget
Foxp3(+) regulatory T (Treg) cells are broadly divided into naive-like and activated Treg cells, however recent studies suggest further Treg cell heterogeneity. Treg cells contribute to impaired T cell responses in chronic infections, but the role of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f151a0dc820858f214515f1145d92b10
http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-145165
http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-145165
NO Is a Macrophage Autonomous Modifier of the Cytokine Response to Streptococcal Single-Stranded RNA
Autor:
Shizuo Akira, Sachin D. Deshmukh, Julia Wennekamp, Katrin Hese, Douglas T. Golenbock, Katharina S. Rauch, Sabrina Müller, Philipp Henneke, Osamu Takeuchi
Publikováno v:
The Journal of Immunology. 188:774-780
Group B streptococci, a major cause of sepsis, induce inflammatory cytokines in strict dependence on bacterial ssRNA and the host molecules MyD88 and UNC-93B. In this study, we show that NO plays an important role in Group B streptococci-induced tran
Autor:
Anne Schönle, Gabriele Prinz, Anne Kathrin Hechinger, Frederike A. Hartl, Kerstin Fehrenbach, Keli Hippen, Jan Mentzel, Justus Duyster, Sebastian A. Wohlfeil, Susana Minguet, Ann Katrin Ruess, Dietmar Pfeifer, Robert Zeiser, Annette Schmitt-Graeff, Miguel A. del Pozo, Theresa Nöltner, Wolfgang Melchinger, Alessandro Prestipino, Katharina S. Rauch, Petya Apostolova, Marta C. Guadamillas, Marie Follo, Kristina Schachtrup, Bruce R. Blazar
Publikováno v:
Blood
Caveolin-1 (Cav-1) is a key organizer of membrane specializations and a scaffold protein that regulates signaling in multiple cell types. We found increased Cav-1 expression in human and murine T cells after allogeneic hematopoietic cell transplantat