Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Katharina Pfeiffer"'
Autor:
Jan Pennig, Katharina Pfeiffer, Natalie Hoppe, Andreas Zirlik, C Haerdtner, Mark Colin Gissler, Philipp Scherrer, Florian Willecke, N Machulsky, C. Bode, Ingo Hilgendorf, Dennis Wolf, Peter Stachon, N. Anto Michel, D. von Elverfeldt
Publikováno v:
European Heart Journal. 40
Introduction Many clinical and experimental observations have established an association between visceral obesity and chronic adipose tissue inflammation. Potent pro-inflammatory mediators such as TNFα, CD40 and IL-1β are regulated by Tumor necrosi
Autor:
Dennis Wolf, Natalie Hoppe, Florian Willecke, Timoteo Marchini, Roland Schüle, Akula Bala Pramod, Timo Heidt, Peter Stachon, Ingo Hilgendorf, Christoph Bode, Andreas Zirlik, Christian Colberg, Konrad Buscher, Erik Ehinger, Ulrich Kintscher, Katharina Pfeiffer, Dietmar Pfeifer, Nathaly Anto Michel, Sebastian Brachs, Klaus Ley, Bianca Dufner, Constantin von zur Mühlen, Dominik von Elverfeldt, Björn Sommer
Rationale: The coincidence of inflammation and metabolic derangements in obese adipose tissue has sparked the concept of met-inflammation. Previous observations, however, suggest that inflammatory pathways may not ultimately cause dysmetabolism. Obje
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd8f64e995dee8cf3a802b738ed3f2cd
https://www.ahajournals.org/doi/10.1161/CIRCRESAHA.117.312055
https://www.ahajournals.org/doi/10.1161/CIRCRESAHA.117.312055
Autor:
Miriam Kalbitz, Tim Eiseler, Florian Gebhard, David A. C. Messerer, Holger Barth, Rebecca Wiegner, Stephanie Denk, Manfred Weiss, John D. Lambris, Stephan Paschke, Ronald P. Taylor, Eberhard Barth, Markus Huber-Lang, Tobias Martin, Katharina Pfeiffer, Erika M. Cook, Margaret A. Lindorfer
The complement and neutrophil defence systems, as major components of innate immunity, are activated during inflammation and infection. For neutrophil migration to the inflamed region, we hypothesized that the complement activation product C5a induce
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cbc97bddaa20246781c22d632270e241
https://europepmc.org/articles/PMC5773343/
https://europepmc.org/articles/PMC5773343/
Autor:
C. Bode, Dennis Wolf, N. Anto Michel, Katharina Pfeiffer, Esther Lutgens, Mark Colin Gissler, Ingo Hilgendorf, Norbert Gerdes, C. Haertner, Florian Willecke, Lesca M. Holdt, Peter Stachon, Andreas Zirlik
Publikováno v:
European Heart Journal. 38
Autor:
Lesley-Ann Huggins, Andreas Zirlik, Mark J. Graham, Ira J. Goldberg, Florian Willecke, Adam E. Mullick, Edward A. Fisher, Katharina Pfeiffer, Tomasz Wietecha, Debapriya Basu, Allison Mogul, Shelley Barnhart, Yunying Hu, Karin E. Bornfeldt
Publikováno v:
Circulation research. 122(4)
Rationale: Animal models have been used to explore factors that regulate atherosclerosis. More recently, they have been used to study the factors that promote loss of macrophages and reduction in lesion size after lowering of plasma cholesterol level
Autor:
Katharina Pfeiffer, Carmen Härtner, Mark Colin Gissler, Ingo Hilgendorf, Christoph Bode, Dennis Wolf, Norbert Gerdes, Nathaly Anto Michel, Lesca M. Holdt, Esther Lutgens, Andreas Zirlik, Florian Willecke, Peter Stachon
Publikováno v:
Atherosclerosis. 263:e62-e63
Autor:
N. Anto-Michel, A. Mullick, Natalie Hoppe, Florian Willecke, Andreas Zirlik, Katharina Pfeiffer, Mark Colin Gissler
Publikováno v:
Atherosclerosis. 275:e49
Autor:
Gunnar Mellgren, Dennis Wolf, Timoteo Marchini, Nathaly Anto-Michel, Katharina Pfeiffer, Lisa Spiga, Christian Smolka, Christoph Koentges, Carmen Härdtner, Timothy Mwinyella, Jan Pennig, Steffen U. Eisenhardt, Andreas Zirlik, Dominik von Elverfeldt, Xiaowei Li, Sven Piepenburg, Peter Stachon, Simon N. Dankel, Constantin von zur Mühlen, Natalie Hoppe, Florian Willecke, Jan-Inge Bjune, Ingo Hilgendorf, Christoph Bode, Timo Heidt, Philipp Scherrer, Tijani Abogunloko, Heiko Bugger, Mark Colin Gissler, Lucia Sol Mitre, Bianca Dufner, Gabriel Seifert
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology
Objective: The accumulation of inflammatory leukocytes is a prerequisite of adipose tissue inflammation during cardiometabolic disease. We previously reported that a genetic deficiency of the intracellular signaling adaptor TRAF5 (TNF [tumor necrosis