Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Katelyn J. Hoff"'
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 16 (2022)
Heterozygous, missense mutations in both α- and β-tubulin genes have been linked to an array of neurodevelopment disorders, commonly referred to as “tubulinopathies.” To date, tubulinopathy mutations have been identified in three β-tubulin iso
Externí odkaz:
https://doaj.org/article/adc643f3f58e4cf49297aed402d27b2e
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 9 (2022)
Developing neurons undergo dramatic morphological changes to appropriately migrate and extend axons to make synaptic connections. The microtubule cytoskeleton, made of α/β-tubulin dimers, drives neurite outgrowth, promotes neuronal growth cone resp
Externí odkaz:
https://doaj.org/article/a936175a0bd84d2ea8ce56d2863b7c90
Autor:
Kristen Park, Katelyn J. Hoff, Linnea Wethekam, Nicholas Stence, Margarita Saenz, Jeffrey K. Moore
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 9 (2021)
Mutations in the family of genes encoding the tubulin subunits of microtubules are associated with a spectrum of human brain malformations known as tubulinopathies. How these mutations impact tubulin activity to give rise to distinct developmental co
Externí odkaz:
https://doaj.org/article/ac8d754a64604c65ac813b97bc4abbe2
Publikováno v:
eLife, Vol 11 (2022)
Heterozygous, missense mutations in α- or β-tubulin genes are associated with a wide range of human brain malformations, known as tubulinopathies. We seek to understand whether a mutation’s impact at the molecular and cellular levels scale with t
Externí odkaz:
https://doaj.org/article/8211b609b1274c8a87b301f2eff5db52
Autor:
Madison M. Rose, Veronica L. Espinoza, Katelyn J. Hoff, Laura A. Pike, Vibha Sharma, Marie-Claude Hofmann, Aik Choon Tan, Nikita Pozdeyev, Rebecca E. Schweppe
Publikováno v:
Cancers; Volume 15; Issue 2; Pages: 378
Patients with advanced thyroid cancer, including advanced papillary thyroid cancer and anaplastic thyroid cancer (ATC), have low survival rates because of the lack of efficient therapies available that can combat their aggressiveness. A total of 90%
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46f239aa81b071eb67a81608b60dc1c8
Heterozygous, missense mutations in α- or β-tubulin genes are associated with a wide range of human brain malformations, known as tubulinopathies. We seek to understand whether a mutation’s impact at the molecular and cellular levels scale with t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5dca25a1a7c0141f2c2eb0666e8a9f2c
https://doi.org/10.1101/2021.12.06.471490
https://doi.org/10.1101/2021.12.06.471490
Autor:
Jeffrey K. Moore, Linnea Wethekam, Nicholas Stence, Margarita Saenz, Katelyn J. Hoff, Kristen Park
Publikováno v:
Frontiers in Cell and Developmental Biology
Frontiers in Cell and Developmental Biology, Vol 9 (2021)
Frontiers in Cell and Developmental Biology, Vol 9 (2021)
Mutations in the family of genes encoding the tubulin subunits of microtubules are associated with a spectrum of human brain malformations known as tubulinopathies. How these mutations impact tubulin activity to give rise to distinct developmental co
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e58d1ca1d7c01bf5d216399943aafa38
http://europepmc.org/articles/PMC8637541
http://europepmc.org/articles/PMC8637541
Autor:
Bogdan Czerniak, Ana Chauca-Diaz, Katelyn J Hoff, Dan Theodorescu, Robert T. Jones, Jason E. Duex, James C. Costello, Annie Jean, Jonathan Kaye, Hany A Abdel-Hafiz, Megan M. Tu, Beth A. Jirón Tamburini, Garrett M. Dancik
Publikováno v:
Communications Biology
Immunotherapies targeting the PD-1/PD-L1 axis are now a mainstay in the clinical management of multiple cancer types, however, many tumors still fail to respond. CCL2 is highly expressed in various cancer types and has been shown to be associated wit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1485d0503c12d183f7c3226589fa361c
https://doi.org/10.1038/s42003-020-01441-y
https://doi.org/10.1038/s42003-020-01441-y