Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Katarzyna Kaziród"'
Autor:
Paulina Podkalicka, Olga Mucha, Katarzyna Kaziród, Krzysztof Szade, Jacek Stępniewski, Liudmyla Ivanishchuk, Hirofumi Hirao, Ewelina Pośpiech, Alicja Józkowicz, Jerzy W. Kupiec-Weglinski, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-19 (2022)
Abstract Although Duchenne muscular dystrophy (DMD) primarily affects muscle tissues, the alterations to systemic metabolism manifested in DMD patients contribute to the severe phenotype of this fatal disorder. We propose that microRNA-378a (miR-378)
Externí odkaz:
https://doaj.org/article/31f8767de0804f9ea3cdfb34755154d9
Autor:
Olga Mucha, Paulina Podkalicka, Katarzyna Kaziród, Emilia Samborowska, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Skeletal Muscle, Vol 11, Iss 1, Pp 1-16 (2021)
Abstract Background Duchenne muscular dystrophy (DMD) is an incurable disease, caused by the mutations in the DMD gene, encoding dystrophin, an actin-binding cytoskeletal protein. Lack of functional dystrophin results in muscle weakness, degeneration
Externí odkaz:
https://doaj.org/article/60466cde0c564a7bb16d8020398b93f5
Autor:
Paulina Podkalicka, Olga Mucha, Katarzyna Kaziród, Iwona Bronisz-Budzyńska, Sophie Ostrowska-Paton, Mateusz Tomczyk, Kalina Andrysiak, Jacek Stępniewski, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Biomedicines, Vol 9, Iss 5, p 481 (2021)
Duchenne muscular dystrophy (DMD), caused by a lack of functional dystrophin, is characterized by progressive muscle degeneration. Interestingly, dystrophin is also expressed in endothelial cells (ECs), and insufficient angiogenesis has already been
Externí odkaz:
https://doaj.org/article/3c0bb1db19374aebb98eca93a846744d
Autor:
Paulina Podkalicka, Katarzyna Kaziród, Jozef Dulak, Agnieszka Łoboda, Emilia Samborowska, Olga Mucha
Publikováno v:
Skeletal Muscle, Vol 11, Iss 1, Pp 1-16 (2021)
Skeletal Muscle
Skeletal Muscle
Background Duchenne muscular dystrophy (DMD) is an incurable disease, caused by the mutations in the DMD gene, encoding dystrophin, an actin-binding cytoskeletal protein. Lack of functional dystrophin results in muscle weakness, degeneration, and as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b5e171765a4c7928bde75e7186b03521
https://doaj.org/article/60466cde0c564a7bb16d8020398b93f5
https://doaj.org/article/60466cde0c564a7bb16d8020398b93f5
Publikováno v:
Cellular and molecular life sciences : CMLS. 79(12)
Hydrogen sulfide (H2S) has been known for years as a poisoning gas and until recently evoked mostly negative associations. However, the discovery of its gasotransmitter functions suggested its contribution to various physiological and pathological pr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63394fd1563fa001fb8ddb7e15d8ac89
https://pubmed.ncbi.nlm.nih.gov/36441348
https://pubmed.ncbi.nlm.nih.gov/36441348
Autor:
Olga Mucha, Katarzyna Kaziród, Paulina Podkalicka, Kinga Rusin, Józef Dulak, Agnieszka Łoboda
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 1; Pages: 470
International Journal of Molecular Sciences, Vol 23, Iss 470, p 470 (2022)
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 23, Iss 470, p 470 (2022)
International Journal of Molecular Sciences
Dysregulation of autophagy may contribute to the progression of various muscle diseases, including Duchenne muscular dystrophy (DMD). Heme oxygenase-1 (HO-1, encoded by Hmox1), a heme-degrading enzyme, may alleviate symptoms of DMD, inter alia, throu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e12f05b0a39e8de46b794356f7024fec
Autor:
Paulina Podkalicka, Olga Mucha, Katarzyna Kaziród, Krzysztof Szade, Jacek Stępniewski, Liudmyla Ivanishchuk, Hirofumi Hirao, Ewelina Pośpiech, Alicja Józkowicz, Jerzy W. Kupiec-Weglinski, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Scientific reports. 12(1)
Although Duchenne muscular dystrophy (DMD) primarily affects muscle tissues, the alterations to systemic metabolism manifested in DMD patients contribute to the severe phenotype of this fatal disorder. We propose that microRNA-378a (miR-378) alters c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bffbc72d290d8c077c9a855d70cfaa2a
https://pubmed.ncbi.nlm.nih.gov/35273230
https://pubmed.ncbi.nlm.nih.gov/35273230
Autor:
Katarzyna Kaziród, Olga Mucha, Paulina Podkalicka, Jozef Dulak, Iwona Bronisz-Budzyńska, Agnieszka Łoboda, Kalina Andrysiak, Sophie Ostrowska-Paton, Jacek Stępniewski, Mateusz Tomczyk
Publikováno v:
Biomedicines
Volume 9
Issue 5
Biomedicines, Vol 9, Iss 481, p 481 (2021)
Volume 9
Issue 5
Biomedicines, Vol 9, Iss 481, p 481 (2021)
Duchenne muscular dystrophy (DMD), caused by a lack of functional dystrophin, is characterized by progressive muscle degeneration. Interestingly, dystrophin is also expressed in endothelial cells (ECs), and insufficient angiogenesis has already been
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b2b4841db4bc09f906bfcb14409bf0ac
Background: Duchenne muscular dystrophy (DMD) is an incurable disease, caused by the mutations in the DMD gene, encoding dystrophin, an actin-binding cytoskeletal protein. Lack of functional dystrophin results in muscle weakness, degeneration, and as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a2cb6d8e7dd65930e65e33440ae532a8
https://doi.org/10.21203/rs.3.rs-140320/v1
https://doi.org/10.21203/rs.3.rs-140320/v1
