Zobrazeno 1 - 10
of 24
pro vyhledávání: '"Katariina Kainulainen"'
Publikováno v:
Antimicrobial Stewardship & Healthcare Epidemiology, Vol 3 (2023)
Abstract Objective: Hospital-acquired (HA) COVID-19 infections are known to increase morbidity and mortality. The aim of this study was to investigate the incidence and outcome of HA COVID-19 in different specialties across the wards in 18 hospitals
Externí odkaz:
https://doaj.org/article/1dbed698676d4943bd88142775d58299
Autor:
Tuomo Polvikoski, Petteri Viramo, Katariina Kainulainen, Raimo Sulkava, Kati Juva, Auli Verkkoniemi, Kimmo Kontula
Publikováno v:
International Psychogeriatrics. 12:379-387
We examined 510 subjects representing 83.2% of all citizens of a Finnish city aged 85 years or over. Mini-Mental State Examination (MMSE) scores, diagnosis of dementia by DSM-III-R criteria, and Apo-E genotype were determined. The prevalence of demen
Autor:
Kirsi Piippo, Pekka Keto, Heikki Swan, Päivi Heikkilä, Katariina Kainulainen, Lauri Toivonen, Juha Kere, Matti Viitasalo, Timo Paavonen, Kimmo Kontula
Publikováno v:
Journal of the American College of Cardiology. 34(7):2035-2042
OBJECTIVESThe purpose of this study was to provide clinical and anatomical characteristics as well as genetic background of a malignant arrhythmogenic disorder.BACKGROUNDAn inherited autosomally dominant cardiac syndrome causing stress-induced polymo
Autor:
Anu Stolt, Matti Viitasalo, Jouko Karjalainen, Matti Mäntysaari, Kimmo Kontula, Urho M. Kujala, Katariina Kainulainen
Publikováno v:
Journal of the American College of Cardiology. 34:494-499
OBJECTIVES We studied whether left ventricular mass in athletes associates with polymorphisms in genes encoding components of the renin–angiotensin system. BACKGROUND Adaptive left ventricular hypertrophy is a feature of the athlete’s heart. Howe
Publikováno v:
Human Mutation. 11:S34-S37
Two novel fibrillin-1 mutations resulting in premature termination codonsbut in different mutant transcript levels and clinical phenotypes.
Autor:
Matti Viitasalo, Kirsi Saarinen, Kimmo Kontula, Katariina Kainulainen, Heikki Swan, Lauri Toivonen
Publikováno v:
Human Mutation. 11:158-165
At least three different gene loci were recently shown to account for the long QT syndrome (LQTS), a monogenic disorder with altered myocardial repolarization and occurrence of life-threatening cardiac arrhythmias. We screened 44 unrelated probands f
Publikováno v:
European Journal of Human Genetics. 2:66-75
The diagnosis of Marfan syndrome (MFS) is still based on careful clinical examination. There are, however, many factors creating problems in the firm establishment of the correct diagnosis. After the identification of the defective gene in MFS, fibri
Mutations in the fibrillin gene responsible for dominant ectopia lentis and neonatal Marfan syndrome
Publikováno v:
Nature Genetics. 6:64-69
Mutations in the gene coding for fibrillin on chromosome 15 (FBN1) are known to cause Marfan syndrome (MFS). A related disorder, dominant ectopia lentis (EL), has also been linked genetically to this locus. We now describe ten novel mutations of FBN1
Autor:
Heli Siikamäki, Pia Kivelä, Pyry N Sipilä, Annikaisa Kettunen, Jukka Ollgren, Anu Kantele, Katariina Kainulainen
Publikováno v:
Journal of travel medicine. 18(4)
Background. Returning travelers with fever pose challenges for clinicians because of the multitude of diagnostic alternatives. Case data in a Finnish tertiary hospital were analyzed in order to define the causes of fever in returned travelers and to
Autor:
Leena Taittonen, Mika Kähönen, Nina Hutri-Kähönen, Jorma Viikari, Katariina Kainulainen, Helena E. Miettinen, Olli T. Raitakari, Md. Shaheenul Islam, Kimmo Kontula, Terho Lehtimäki, Markus Juonala
Publikováno v:
Atherosclerosis. 188(2)
The roles of angiotensin converting enzyme (ACE) insertion–deletion (I/D) and angiotensinogen (AGT) m235t polymorphisms in cardiovascular diseases have been investigated extensively during the past decade but results have been inconsistent. A sex-s