Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Katalin Éva, Sikura"'
Autor:
Zsolt Combi, László Potor, Péter Nagy, Katalin Éva Sikura, Tamás Ditrói, Eszter Petra Jurányi, Klaudia Galambos, Tamás Szerafin, Péter Gergely, Matthew Whiteman, Roberta Torregrossa, Yuchao Ding, Lívia Beke, Zoltán Hendrik, Gábor Méhes, György Balla, József Balla
Publikováno v:
Redox Biology, Vol 60, Iss , Pp 102629- (2023)
Hydrogen sulfide (H2S) was previously revealed to inhibit osteoblastic differentiation of valvular interstitial cells (VICs), a pathological feature in calcific aortic valve disease (CAVD). This study aimed to explore the metabolic control of H2S lev
Externí odkaz:
https://doaj.org/article/a3796fa1603843a4bad5792c6dc90283
Autor:
Katalin Éva Sikura, Zsolt Combi, László Potor, Tamás Szerafin, Zoltán Hendrik, Gábor Méhes, Péter Gergely, Matthew Whiteman, Lívia Beke, Ibolya Fürtös, György Balla, József Balla
Publikováno v:
Journal of Advanced Research, Vol 27, Iss , Pp 165-176 (2021)
Introduction: Hydrogen sulfide (H2S) was revealed to inhibit aortic valve calcification and inflammation was implicated in the pathogenesis of calcific aortic valve disease (CAVD). Objectives: We investigate whether H2S inhibits mineralization via ab
Externí odkaz:
https://doaj.org/article/bebf49e1eaec4503b17f19f754ce6c93
Autor:
László Potor, József Balla, Katalin Éva Sikura, Ibolya Fürtös, Zoltán Hendrik, Zsolt Combi, Péter Gergely, György Balla, Matthew Whiteman, Gábor Méhes, Tamás Szerafin, Lívia Beke
Publikováno v:
Journal of Advanced Research
Journal of Advanced Research, Vol 27, Iss, Pp 165-176 (2021)
Journal of Advanced Research, Vol 27, Iss, Pp 165-176 (2021)
Graphical abstract
Highlights • H2S derived from CSE/CBS inhibits inflammation and calcification of valvular interstitial cells. • H2S releasing molecules inhibits inflammation and calcification of valvular interstitial cells. • H2S inhibi
Highlights • H2S derived from CSE/CBS inhibits inflammation and calcification of valvular interstitial cells. • H2S releasing molecules inhibits inflammation and calcification of valvular interstitial cells. • H2S inhibi
Autor:
Melinda Oros, Csaba Tóth, József Posta, Niké Posta, József Balla, Katalin Éva Sikura, Zoltán Hendrik, Gergő Kalló, Gábor Méhes, György Balla, Éva Csősz, Dávid Pethő, László Potor
Publikováno v:
Laboratory Investigation; a Journal of Technical Methods and Pathology
The lysis of red blood cells was shown to occur in human ruptured atherosclerotic lesions and intraventricular hemorrhage (IVH) of the brain. Liberated cell-free hemoglobin was found to undergo oxidation in both pathologies. We hypothesize that hemog
Oxidation of Hemoglobin Drives a Proatherogenic Polarization of Macrophages in Human Atherosclerosis
Autor:
László, Potor, Zoltán, Hendrik, Andreas, Patsalos, Éva, Katona, Gábor, Méhes, Szilárd, Póliska, Éva, Csősz, Gergő, Kalló, István, Komáromi, Zsolt, Combi, Niké, Posta, Katalin Éva, Sikura, Dávid, Pethő, Melinda, Oros, György, Vereb, Csaba, Tóth, Péter, Gergely, László, Nagy, György, Balla, József, Balla
Publikováno v:
Antioxidantsredox signaling. 35(12)
Autor:
Maura Poli, Abolfazl Zarjou, Niké Posta, Tamás Szerafin, Lívia Beke, László Potor, József Balla, György Balla, Anupam Agarwal, Melinda Oros, Gábor Méhes, Zoltán Hendrik, Katalin Éva Sikura, Ibolya Fürtös, Paolo Arosio
Objective— Calcific aortic valve disease is a prominent finding in elderly and in patients with chronic kidney disease. We investigated the potential role of iron metabolism in the pathogenesis of calcific aortic valve disease. Approach and Results
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::475ce3d3cf9180eea56ec5351b021d64
Autor:
Katalin Éva, Sikura, László, Potor, Tamás, Szerafin, Melinda, Oros, Péter, Nagy, Gábor, Méhes, Zoltán, Hendrik, Abolfazl, Zarjou, Anupam, Agarwal, Niké, Posta, Roberta, Torregrossa, Matthew, Whiteman, Ibolya, Fürtös, György, Balla, József, Balla
Publikováno v:
British Journal of Pharmacology
Background and Purpose Calcification of heart valves is a frequent pathological finding in chronic kidney disease and in elderly patients. Hydrogen sulfide (H2S) may exert anti‐calcific actions. Here we investigated H2S as an inhibitor of valvular