Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Karla Posch"'
Publikováno v:
Diabetes Research and Clinical Practice. 45:153-160
In diabetes mellitus, the risk for cardiovascular complications and development of atherosclerosis is increased compared with healthy individuals. Recently evidence was provided that increased production of superoxide anions occurs in endothelial cel
Publikováno v:
Europe PubMed Central
The effects of acute hyperglycemia on endothelial Ca2+ signaling, formation of endothelium-derived relaxing factor (EDRF) and bioactivity of EDRF were investigated. Hyperglycemia increased 2,5-tert-butyl-1,4-hydrochinone (BHQ)-initiated Ca2+ signalin
Autor:
Thomas C. Wascher, Wolfgang F. Graier, Sandra Wallner, Albin Hermetter, Gert M. Kostner, Karla Posch
Publikováno v:
Biochemical and Biophysical Research Communications. 234:35-38
L-arginine supplementation is hypothesized to reduce endothelial dysfunction and atherogenesis via increased biosynthesis of nitric oxide. Here we describe superoxide scavenging properties of arginine as an additional aspect which needs to be conside
Autor:
T. C. Wascher, Günther Jürgens, Wolfgang F. Graier, Sabine Simecek, Gert M. Kostner, Karla Posch, Sabina Baumgartner-Parzer
Publikováno v:
Diabetes. 48(6)
Little is known about the mechanism(s) of endothelial dysfunction in diabetes. In this study, the effect of nonenzymatic glycated LDL, a phenomenon induced by elevated D-glucose levels associated with diabetes, on porcine aortic endothelial cells was
Publikováno v:
Life sciences. 64(8)
The aim of this study was to investigate the kinetics of L-arginine transport mechanisms and the role of extracellular L-arginine in nitric oxide formation during shear stress activation of endothelial cells. Porcine aortic endothelial cells were gro
Publikováno v:
The Journal of physiology. 513
1. The involvement of ryanodine-sensitive Ca2+ release (RsCR) in bradykinin (Bk)-induced Ca2+ release, capacitative Ca2+ entry (CCE) and nitric oxide synthase (NOS) activation was assessed in freshly isolated bovine coronary artery endothelial cells.
Publikováno v:
Atherosclerosis. 130:S2