Zobrazeno 1 - 10
of 67
pro vyhledávání: '"Karl M Koch"'
Autor:
Karl M. Koch, Walter Entires, Reinhard Brunkhorst, Hans L. Tillmann, W. Stangel, Volker Kliem, Dong-Feng Chen, Michael P. Manns
Publikováno v:
Liver. 16:384-389
In hepatitis C, both susceptibility to infection and the course of disease may depend on differences in the immune response. As the major histocompatibility complex (MHC) plays a crucial role in antigen presentation, we investigated a possible relati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6230f07529beb4c7f875bd5ccb4d4a50
https://doi.org/10.1111/j.1600-0676.1996.tb00767.x
https://doi.org/10.1111/j.1600-0676.1996.tb00767.x
Autor:
Gerhard Lonnemann, Karl M. Koch
Publikováno v:
Journal of the American Society of Nephrology. 13:S72-S77
The available data on the pathophysiology of beta(2)-microglobulin amyloidosis (beta(2)mA) suggest that this progressive disease associated with end-stage renal failure develops in several consecutive phases. First, declining kidney function leads to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cf8e03fa09fe3f88d22c8ad9ba6bd965
https://doi.org/10.1681/asn.v13suppl_1s72
https://doi.org/10.1681/asn.v13suppl_1s72
Autor:
Gerhard Lonnemann, Karl M. Koch
Publikováno v:
Nephrology Dialysis Transplantation. 16:17-22
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6b5dc3355181d6600edb676dd073b84a
https://doi.org/10.1093/ndt/16.suppl_4.17
https://doi.org/10.1093/ndt/16.suppl_4.17
Publikováno v:
Nephrology Dialysis Transplantation. 16:12-16
beta2-Microglobulin-derived amyloidosis (Abeta2m) represents a major cause or morbidity in patients with end-stage renal disease. Symptoms of Abeta2m amyloid are mainly related to (peri-) articular amyloid deposition. Conventional non-invasive diagno
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be0349b73a09750860969332dd44fa0c
https://doi.org/10.1093/ndt/16.suppl_4.12
https://doi.org/10.1093/ndt/16.suppl_4.12