Zobrazeno 1 - 10
of 48
pro vyhledávání: '"Karl Heinrich Fritzemeier"'
Autor:
Astrid Menning, Alexander Walter, Marion Rudolph, Isabella Gashaw, Karl-Heinrich Fritzemeier, Lars Roese
Publikováno v:
PLoS ONE, Vol 7, Iss 8, p e41800 (2012)
Menstruation-associated disorders negatively interfere with the quality of life of many women. However, mechanisms underlying pathogenesis of menstrual disorders remain poorly investigated up to date. Among others, this is based on a lack of appropri
Externí odkaz:
https://doaj.org/article/0ec7db3ab32d4489b40c5e92af9a65dd
Autor:
Frank Hülsemann, Carmen Weigt, Patrick Diel, T. Hertrampf, Pinar Kurnaz, Ulrich Flenker, Karl Heinrich Fritzemeier
Publikováno v:
The Journal of Steroid Biochemistry and Molecular Biology. 154:12-22
The leptin resistant Zucker diabetic fatty (ZDF) rats are hyperphagic and become obese, but whereas the males develop type 2 diabetes mellitus (T2DM), the females remain euglycaemic. As estrogen deficiency is known to increase the risk of developing
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c578d78a13a78d86251fa04fe6a52912
https://doi.org/10.1016/j.jsbmb.2015.06.002
https://doi.org/10.1016/j.jsbmb.2015.06.002
Autor:
Ulrich Flenker, Patrick Diel, T. Hertrampf, Felix M. Kluxen, Frank Hülsemann, Carmen Weigt, Karl Heinrich Fritzemeier
Publikováno v:
Molecular and Cellular Endocrinology. 377:147-158
The molecular mechanisms underlying the effects of selective ER subtype activation on lipogenesis, adipogenesis, lipid utilization and storage as well as glucose metabolism are currently largely unknown and were analyzed in female OVX Wistar rats on
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9582cb833ede0c1567ac9ad7c5ed4ae5
https://doi.org/10.1016/j.mce.2013.07.007
https://doi.org/10.1016/j.mce.2013.07.007
Publikováno v:
Biochemical and Biophysical Research Communications. 423:850-856
Activation of vascular mineralocorticoid (MR) or estrogen receptors (ER) exerts opposing effects on vascular remodeling. As we have previously shown, activation of either estrogen receptor subtype, ERα or ERβ, is fully sufficient to attenuate vascu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c9511b93f758429fd5ebe86923957a5e
https://doi.org/10.1016/j.bbrc.2012.06.053
https://doi.org/10.1016/j.bbrc.2012.06.053
Autor:
M. Velders, Patrick Diel, Felix M. Kluxen, T. Hertrampf, B. Schleipen, Karl Heinrich Fritzemeier, A Lorenz, Almut F. Molzberger
Publikováno v:
Carcinogenesis. 32:1675-1683
Epidemiological data indicate that intake of estrogens and isoflavones may be beneficial for the prevention of colorectal cancer (CRC). Based on this data, the aim of the study was to investigate estrogen receptor (ER) subtype-specific effects on int
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::52e27f0d35c578cedac148c3b36a6179
https://doi.org/10.1093/carcin/bgr188
https://doi.org/10.1093/carcin/bgr188
Autor:
Devi Ngo, Evan R. Simpson, Yuan Hang Yang, Eric F Morand, Margaret E. E. Jones, Karl Heinrich Fritzemeier
Publikováno v:
Arthritis & Rheumatism. 62:1017-1025
Objective A number of experimental observations have associated elevated estrogen levels with amelioration of inflammation. The involvement of estrogen and estrogen receptor (ER) isotypes in the regulation of inflammation in males is not well underst
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d690fd4cdce06452b64f0387e581e3d
https://doi.org/10.1002/art.27330
https://doi.org/10.1002/art.27330
Autor:
Patrick Diel, B. Schleipen, T. Hertrampf, M. Velders, U. Laudenbach, Karl Heinrich Fritzemeier
Publikováno v:
Molecular and Cellular Endocrinology
Molecular and Cellular Endocrinology, Elsevier, 2008, 291 (1-2), pp.104. ⟨10.1016/j.mce.2008.03.003⟩
Molecular and Cellular Endocrinology, Elsevier, 2008, 291 (1-2), pp.104. ⟨10.1016/j.mce.2008.03.003⟩
To further elucidate the processes involved in the physiology of bone-protection by estrogens, ovariectomized (OVX) rats were treated subcutaneously with 17beta-estradiol (E(2)), the ERalpha-specific agonist (16alpha-LE2) and the ERbeta-specific agon
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7e5bcf5b8e1a3513f4ae8c8e07a1acab
https://doi.org/10.1016/j.mce.2008.03.003
https://doi.org/10.1016/j.mce.2008.03.003
Autor:
Katja Prelle, Alexander Walter, Helga Altmann, Karl-Heinrich Fritzemeier, Mario Klewer, Christiane Otto, Iris Fuchs, Richardus Vonk
Publikováno v:
Endocrinology. 149:3952-3959
The role of progestins in combined hormone therapy is the inhibition of uterine epithelial cell proliferation. The Women’s Health Initiative study provided evidence for an increased risk of breast cancer in women treated with conjugated equine estr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be4d9751a3142bf04212e4f698f964ad
https://doi.org/10.1210/en.2007-1612
https://doi.org/10.1210/en.2007-1612
Publikováno v:
British Journal of Pharmacology. 153:1432-1437
Background and purpose: Studies with oestrogen receptorα (ERα)- and ERβ-selective compounds have already shown that the effects of 17β-estradiol (E2) on body weight, movement drive and bone-protection are mediated via ERα. This study was based o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::363c43dca5e53f6a8e94eae545dba98f
https://doi.org/10.1038/sj.bjp.0707664
https://doi.org/10.1038/sj.bjp.0707664
Autor:
Jenny D. Y. Chow, Evan R. Simpson, Rachel Anne Hill, Wah Chin Boon, Karl-Heinrich Fritzemeier
Publikováno v:
Molecular and Cellular Neuroscience. 36:146-157
The aromatase (ArKO) knockout mouse is estrogen deficient. Our previous analysis revealed apoptosis of dopaminergic neurons in the arcuate nucleus (Arc) and medial preoptic area (MPO) of 1-year-old male ArKO mice. We sought to determine which estroge
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::212e1c339199caf9a6be2490391727df
https://doi.org/10.1016/j.mcn.2007.05.012
https://doi.org/10.1016/j.mcn.2007.05.012