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CD4+ T cells regulate sickness-induced anorexia and fat wasting during a chronic parasitic infection
Autor:
Samuel E. Redford, Siva Karthik Varanasi, Karina K. Sanchez, Natalia R. Thorup, Janelle S. Ayres
Publikováno v:
Cell Reports, Vol 42, Iss 8, Pp 112814- (2023)
Summary: Infections cause catabolism of fat and muscle stores. Traditionally, studies have focused on understanding how the innate immune system contributes to energy stores wasting, while the role of the adaptive immune system remains elusive. In th
Externí odkaz:
https://doaj.org/article/e7a521f7f4e0428fa19a3ab7c96f83df
Autor:
Karina K. Sanchez, Katia Troha, Andre Mu, Samuel E. Redford, Justin L. McCarville, Arianna Insenga, Sarah Stengel, Yujung Michelle Lee, Janelle S. Ayres
Publikováno v:
Infect Immun
To combat infections, hosts employ a combination of antagonistic and cooperative defense strategies. The former refers to pathogen killing mediated by resistance mechanisms, while the latter refers to physiological defense mechanisms that promote hos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::456887ea8973bbe2e50d61aa4634d783
https://pubmed.ncbi.nlm.nih.gov/35924898
https://pubmed.ncbi.nlm.nih.gov/35924898
CD4+ T cells regulate sickness-induced anorexia and fat wasting during a chronic parasitic infection
Catabolic responses of lean and fat energy stores are a component of the host response to infection. Cachexia is an extreme catabolic state characterized by unintentional weight loss and muscle loss, that can include fat loss. Whether cachexia plays
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7991a1128a64af307904965c7358a2c3
https://doi.org/10.1101/2022.12.17.520896
https://doi.org/10.1101/2022.12.17.520896
To combat infections, hosts employ a combination of antagonistic and cooperative defense strategies. The former refers to pathogen killing mediated by resistance mechanisms, while the latter refers to physiological defense mechanisms that promote hos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e0b0aa5232eb531fde244d80ea853d67
https://doi.org/10.1101/2020.08.24.264648
https://doi.org/10.1101/2020.08.24.264648
Autor:
Alexandria M. Palaferri Schieber, Grischa Y. Chen, Yujung M. Lee, Mathias Leblanc, Janelle S. Ayres, Karina K. Sanchez, Samuel E. Redford, Maxim N. Shokhirev
Publikováno v:
Cell. 175:146-158.e15
Pathogen virulence exists on a continuum. The strategies that drive symptomatic or asymptomatic infections remain largely unknown. We took advantage of the concept of lethal dose 50 (LD50) to ask which component of individual non-genetic variation be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::66b2337bc797b25c0fbda09a1342ecf1
https://doi.org/10.1016/j.cell.2018.07.016
https://doi.org/10.1016/j.cell.2018.07.016