Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Karina J Alcántara-Solano"'
Autor:
Eduardo Cienfuegos-Pecina, Tannya R. Ibarra-Rivera, Alma L. Saucedo, Luis A. Ramírez-Martínez, Deanna Esquivel-Figueroa, Ixel Domínguez-Vázquez, Karina J. Alcántara-Solano, Diana P. Moreno-Peña, Gabriela Alarcon-Galvan, Diana Raquel Rodríguez-Rodríguez, Liliana Torres-González, Linda E. Muñoz-Espinosa, Edelmiro Pérez-Rodríguez, Paula Cordero-Pérez
Publikováno v:
PeerJ, Vol 8, p e9438 (2020)
Background Ischemia–reperfusion (IR) injury is the main cause of delayed graft function in solid organ transplantation. Hypoxia-inducible factors (HIFs) control the expression of genes related to preconditioning against IR injury. During normoxia,
Externí odkaz:
https://doaj.org/article/73d3e0755459469db4041319b76f585a
Autor:
Eduardo, Cienfuegos-Pecina, Tannya R, Ibarra-Rivera, Alma L, Saucedo, Luis A, Ramírez-Martínez, Deanna, Esquivel-Figueroa, Ixel, Domínguez-Vázquez, Karina J, Alcántara-Solano, Diana P, Moreno-Peña, Gabriela, Alarcon-Galvan, Diana Raquel, Rodríguez-Rodríguez, Liliana, Torres-González, Linda E, Muñoz-Espinosa, Edelmiro, Pérez-Rodríguez, Paula, Cordero-Pérez
Publikováno v:
PeerJ
Background Ischemia–reperfusion (IR) injury is the main cause of delayed graft function in solid organ transplantation. Hypoxia-inducible factors (HIFs) control the expression of genes related to preconditioning against IR injury. During normoxia,
Autor:
Diana Raquel Rodríguez-Rodríguez, Alma L. Saucedo, Linda E. Muñoz-Espinosa, Edelmiro Pérez-Rodríguez, Liliana Torres-González, Eduardo Cienfuegos-Pecina, Gabriela Alarcón-Galván, Deanna Esquivel-Figueroa, Luis A. Ramirez-Martinez, Diana Patricia Moreno-Peña, Paula Cordero-Pérez, Ixel Domínguez-Vázquez, Karina J Alcántara-Solano, Tannya R. Ibarra-Rivera
Publikováno v:
PeerJ, Vol 8, p e9438 (2020)
BackgroundIschemia–reperfusion (IR) injury is the main cause of delayed graft function in solid organ transplantation. Hypoxia-inducible factors (HIFs) control the expression of genes related to preconditioning against IR injury. During normoxia, H