Zobrazeno 1 - 10
of 22
pro vyhledávání: '"Karin Mausner-Fainberg"'
Publikováno v:
Multiple Sclerosis International, Vol 2014 (2014)
Background. The neural stem cells (NSCs) migrate to the damaged sites in multiple sclerosis (MS) and in experimental autoimmune encephalomyelitis (EAE). However, the differentiation into neurons or oligodendrocytes is blocked. Epidermal growth factor
Externí odkaz:
https://doaj.org/article/1f0501c0f0334650ae60340478319235
Autor:
Lior Fuchs, Karin Mausner-Fainberg, Anna Luban, Susanna Eva Asseyer, Maya Golan, Moshe Benhamou, Ilan Volovitz, Keren Regev, Ifat Vigiser, Yoav Piura, Hadar Kolb, Friedemann Paul, Arnon Karni
Publikováno v:
Journal of Neuroimmunology. 371:577936
Connective tissue growth factor (CTGF/CCN2) is a proinflammatory and an oligodendrocyte-differentiating blocking agent. It is found in MS lesions, which raises the possibility of involvement in MS pathogenesis. We found that its CSF and serum levels
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2bf59260d6cbfbeaa4bf7dd0655d6463
https://doi.org/10.1016/j.jneuroim.2022.577936
https://doi.org/10.1016/j.jneuroim.2022.577936
Autor:
Adi Wilf-Yarkoni, Maya Golan, Moshe Benhamou, Arnon Karni, Hadar Kolb, Karin Mausner-Fainberg, Keren Regev, Bassima Ibrahim
Publikováno v:
CNS Drugs. 33:1229-1237
The pathophysiology of multiple sclerosis involves an autoimmune and a neurodegenerative mechanism. Central nervous system-infiltrating immune cells in multiple sclerosis also possess a neuroprotective activity through secretion of neurotrophins, suc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::60ab27432baac5ea35119e0eac06a025
https://doi.org/10.1007/s40263-019-00675-7
https://doi.org/10.1007/s40263-019-00675-7
Autor:
Adi Wilf-Yarkoni, Maya Golan, Moshe Benhamou, Arnon Karni, Karin Mausner-Fainberg, Moran Penn, Smadar Gertel
Publikováno v:
Journal of Neuroimmunology. 327:36-40
Demyelination, axonal loss and failure of tissue repair characterize MS lesions. Bone morphogenetic proteins (BMPs) signaling is associated with remyelination failure. Coco is one of the BMP antagonists. We found reduced Coco serum levels in relapsin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0972a772e3efd5c47fe1de9d97df65dd
https://doi.org/10.1016/j.jneuroim.2019.01.009
https://doi.org/10.1016/j.jneuroim.2019.01.009
Autor:
Uri Danon, Maya Golan, Ehud Marom, Arnon Karni, Moshe Benhamou, Karin Mausner-Fainberg, Nadav Bleich Kimelman
Publikováno v:
Neurotherapeutics
Oligodendrocyte precursor cells (OPCs) are present in demyelinated lesions of multiple sclerosis (MS) patients. However, their differentiation into functional oligodendrocytes is insufficient, and most lesions evolve into nonfunctional astroglial sca
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::522e64301a05105b723d1f228b61ee04
https://europepmc.org/articles/PMC8608985/
https://europepmc.org/articles/PMC8608985/
Autor:
Arnon Karni, Hadar Kolb, Karin Mausner-Fainberg, Ifat Vigiser, Keren Regev, Avivit Krivitsky, Moshe Benhamou, Maya Golan
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 2182, p 2182 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 4
International Journal of Molecular Sciences
Volume 22
Issue 4
The effect of the inflammatory response on regenerative processes in the brain is complex. This complexity is even greater when the cause of the tissue damage is an autoimmune response. Multiple sclerosis (MS) is an immune-mediated disease in which d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15e79a58ede779e59d96b81a7d759a62
https://www.mdpi.com/1422-0067/22/4/2182
https://www.mdpi.com/1422-0067/22/4/2182
Publikováno v:
Journal of Neuroimmunology. 310:120-128
Blockage of bone morphogenetic protein (BMP) signaling is required for differentiation of neurons and oligodendrocytes from neural stem cells (NSCs). Sera of untreated relapsing-remitting multiple sclerosis (RR-MS) patients expressed significantly hi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::93605acfff1eab68be4e62f59fbc7c4e
https://doi.org/10.1016/j.jneuroim.2017.07.008
https://doi.org/10.1016/j.jneuroim.2017.07.008
Autor:
Arnon Karni, Yifat Amir Levy, Adi Vaknin-Dembinsky, Tali Amidror, Keren Regev, Karin Mausner-Fainberg
Publikováno v:
Journal of Neuroimmunology. 278:85-89
Leukemia inhibitory factor (LIF) is known to potentiate the differentiation and survival of neuronal and oligodendrocyte precursors. Systemic therapy with LIF reportedly ameliorated the severity of experimental autoimmune encephalomyelitis and preven
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::605e6b0864f3021a2c5421bed1a97dc9
https://doi.org/10.1016/j.jneuroim.2014.12.010
https://doi.org/10.1016/j.jneuroim.2014.12.010
Publikováno v:
Clinical immunology (Orlando, Fla.). 183
Th-17 type immune response that occurs in multiple sclerosis (MS) is linked to CCR6-CCL20 interaction. We confirmed the dependency on CCR6 in EAE development. Vaccination of mice with hCCL20, but not mCCL20, produced anti-murine CCL20 and ameliorated
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5e893f9178a6ba1fddf4ee2c9c0ddaba
https://pubmed.ncbi.nlm.nih.gov/28939272
https://pubmed.ncbi.nlm.nih.gov/28939272