Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Karin Broennimann"'
Publikováno v:
Viruses, Vol 15, Iss 5, p 1129 (2023)
The COVID-19 pandemic resulted from the global spread of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Since its first appearance in 2019, new SARS-CoV-2 variants of concern (VOCs) have emerged frequently, changing the infection
Externí odkaz:
https://doaj.org/article/7f5a2db4666d4884b6adeaa363e779da
Autor:
Assaf Biran, Nadav Myers, Shirel Steinberger, Julia Adler, Marianna Riutin, Karin Broennimann, Nina Reuven, Yosef Shaul
Publikováno v:
Cells, Vol 11, Iss 20, p 3231 (2022)
The degradation of intrinsically disordered proteins (IDPs) by a non-26S proteasome process does not require proteasomal targeting by polyubiquitin. However, whether and how IDPs are recognized by the non-26S proteasome, including the 20S complex, re
Externí odkaz:
https://doaj.org/article/2f45f75fcdbe4cc29236027ee154f4e1
Publikováno v:
Cells, Vol 11, Iss 14, p 2248 (2022)
The hepatitis B virus (HBV) is one of the smallest but most highly infectious human pathogens. With a DNA genome of only 3.2 kb and only four genes, HBV successfully completes its life cycle by using intricate processes to hijack the host machinery.
Externí odkaz:
https://doaj.org/article/227b70302bcc4ae493b4dffbdb7b9c91
Autor:
Karin Broennimann, Inna Ricardo-Lax, Julia Adler, Eleftherios Michailidis, Ype P. de Jong, Nina Reuven, Yosef Shaul
Publikováno v:
Biomolecules, Vol 11, Iss 12, p 1822 (2021)
DNA viruses require dNTPs for replication and have developed different strategies to increase intracellular dNTP pools. Hepatitis B virus (HBV) infects non-dividing cells in which dNTPs are scarce and the question is how viral replication takes place
Externí odkaz:
https://doaj.org/article/0c6341ba99b446c49279ff1fe5ef0c21
Publikováno v:
Biomolecules, Vol 9, Iss 10, p 584 (2019)
CRISPR/Cas9 is a powerful tool for genome editing in cells and organisms. Nevertheless, introducing directed templated changes by homology-directed repair (HDR) requires the cellular DNA repair machinery, such as the MRN complex (Mre11/Rad50/Nbs1). T
Externí odkaz:
https://doaj.org/article/7d30df76585a4b578a32f25424fd2656